Classification and biological diagnostic of angioedema diseases

被引：4

作者：

Drouet, C. ^{[1
]}

Ponard, D. ^{[2
]}

Monnier, N. ^{[3
]}

Lunardi, J. ^{[3
]}

Bosson, J. -L. ^{[4
]}

机构：

[1] Univ Grenoble 1, CHU Grenoble, Ctr Reference Angioedeme, GREPI TIMC IMAG CNRS 5525, F-38043 Grenoble, France

[2] CHU Grenoble, Ctr Reference Angioedeme, Immunol Lab, F-38043 Grenoble, France

[3] CHU Grenoble, Ctr Reference Angioedeme, Lab Biochim & Genet Mol, F-38043 Grenoble, France

[4] Univ Grenoble 1, CHU Grenoble, CIC Inserm, TIMC IMAG CNRS 5525, F-38043 Grenoble, France

来源：

REVUE FRANCAISE D ALLERGOLOGIE ET D IMMUNOLOGIE CLINIQUE | 2008年 / 48卷 / 06期

关键词：

Angioedema; Kinins; Kininogenase; Kininase; C1; Inhibitor; Complement;

D O I：

10.1016/j.allerg.2008.02.004

中图分类号：

R392 [医学免疫学];

学科分类号：

100102 ;

摘要：

The angioedema disease represents a situation associated with production or accumulation of kinins at the endothelial cell surface. A classification of the different forms of the disease can be proposed upon the bases of the metabolic failure, either from the increased proteolytic activities towards kininogens (decreased control by C1 Inhibitor, escape to this control) or from the kinin accumulation associated with the decreased catabolism. The diagnostic Must describe the failure of the C1 Inhibitor control, the gain of function of the kinin-producing kininogenases or the decreased activities of the proteases needed for kinin catabolism (kininases). (c) 2008 Elsevier Masson SAS.

引用

页码：441 / 446

页数：6

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