Targeting p53 tumor suppressor to induce apoptosis and cell cycle arrest in esophageal cancer cells by novel sugar-cholestanols compounds

被引:2
作者
Faried, A. [1 ]
Faried, L. S. [2 ]
Kato, H. [1 ]
Asao, T. [1 ]
Kuwano, H. [1 ]
Yazawa, S. [3 ]
机构
[1] Gunma Univ, Grad Sch Med, Dept Surg, Maebashi, Gunma 371, Japan
[2] Gunma Univ, Grad Sch Med, Dept Gynecol, Maebashi, Gunma 371, Japan
[3] Otsuka Pharmaceut Co Ltd, Tokushima Res Inst, Tokushima 77101, Japan
来源
EJC SUPPLEMENTS | 2008年 / 6卷 / 09期
关键词
D O I
10.1016/S1359-6349(08)71496-4
中图分类号
R73 [肿瘤学];
学科分类号
100214 ;
摘要
引用
收藏
页码:83 / 83
页数:1
相关论文
共 2 条
[1]  
FARIED A, 2006, SEIKAGAKU
[2]   Chemically synthesized sugar-cholestanols possess a preferential anticancer activity involving promising therapeutic potential against human esophageal cancer [J].
Faried, Ahmad ;
Faried, Leri S. ;
Nakagawa, Takashi ;
Yamauchi, Takahito ;
Kitani, Mami ;
Sasabe, Hiroyuki ;
Nishimura, Toyo ;
Usman, Nurhayat ;
Kato, Hiroyuki ;
Asao, Takayuki ;
Kuwano, Hiroyuki ;
Yazawa, Shin .
CANCER SCIENCE, 2007, 98 (09) :1358-1367