Advances in mechanisms of allergy

被引:57
作者
Bochner, BS
Busse, WW
机构
[1] Johns Hopkins Univ, Sch Med, Div Clin Immunol & Allergy, Dept Med, Baltimore, MD USA
[2] Univ Wisconsin, Sch Med, Div Allergy & Immunol, Dept Med, Madison, WI USA
关键词
asthma; rhinitis; sinusitis; surrogate markers; eosinophils; basophils; mast cells; T cells; epithelial cells; chemokines; cytokines; prostaglandins; leukotrienes; animal models; smooth muscle; pathophysiology;
D O I
10.1016/j.jaci.2004.02.016
中图分类号
R392 [医学免疫学];
学科分类号
100102 [免疫学];
摘要
This review summarizes selected Mechanisms of Allergy articles appearing between 2002 and 2003 in the Journal of Allergy and Clinical Immunology. Articles chosen include those dealing with human airways disease pathophysiology, pharmacology, cell biology, cell recruitment, and genetics, as well as information from allergen challenge models in both human and nonhuman systems. When appropriate, articles from other journals have been included to supplement the topics being presented.
引用
收藏
页码:868 / 875
页数:8
相关论文
共 53 条
[1]
Myosin light chain kinase mediates eosinophil chemotaxis in a mitogen-activated protein kinase-dependent manner [J].
Adachi, T ;
Stafford, S ;
Kayaba, H ;
Chihara, J ;
Alam, R .
JOURNAL OF ALLERGY AND CLINICAL IMMUNOLOGY, 2003, 111 (01) :113-116
[2]
The eosinophil - Quo vadis? [J].
Alam, R ;
Busse, WW .
JOURNAL OF ALLERGY AND CLINICAL IMMUNOLOGY, 2004, 113 (01) :38-42
[3]
Imbalance between vascular endothelial growth factor and endostatin levels in induced sputum from asthmatic subjects [J].
Asai, K ;
Kanazawa, H ;
Otani, K ;
Shiraishi, S ;
Hirata, K ;
Yoshikawa, J .
JOURNAL OF ALLERGY AND CLINICAL IMMUNOLOGY, 2002, 110 (04) :571-575
[4]
Verdict in the case of therapies versus eosinophils: The jury is still out [J].
Bochner, BS .
JOURNAL OF ALLERGY AND CLINICAL IMMUNOLOGY, 2004, 113 (01) :3-9
[5]
Release of both CCR4-active and CXCR3-active chemokines during human allergic pulmonary late-phase reactions [J].
Bochner, BS ;
Hudson, SA ;
Xiao, HQ ;
Liu, MC .
JOURNAL OF ALLERGY AND CLINICAL IMMUNOLOGY, 2003, 112 (05) :930-934
[6]
Airway remodeling-associated mediators in moderate to severe asthma:: Effect of steroids on TGF-β, IL-11, IL-17, and type I and type III collagen expression [J].
Chakir, J ;
Shannon, J ;
Molet, S ;
Fukakusa, M ;
Elias, J ;
Laviolette, M ;
Boulet, LP ;
Hamid, Q .
JOURNAL OF ALLERGY AND CLINICAL IMMUNOLOGY, 2003, 111 (06) :1293-1298
[7]
IFN-α inhibits IL-3 priming of human basophil cytokine secretion but not leukotriene C4 and histamine release [J].
Chen, YH ;
Bieneman, AP ;
Creticos, PS ;
Chichester, KL ;
Schroeder, JT .
JOURNAL OF ALLERGY AND CLINICAL IMMUNOLOGY, 2003, 112 (05) :944-950
[8]
Evidence for gene-environment interactions in a linkage study of asthma and smoking exposure [J].
Colilla, S ;
Nicolae, D ;
Pluzhnikov, A ;
Blumenthal, MN ;
Beaty, TH ;
Bleecker, ER ;
Lange, EM ;
Rich, SS ;
Meyers, DA ;
Ober, C ;
Cox, NJ .
JOURNAL OF ALLERGY AND CLINICAL IMMUNOLOGY, 2003, 111 (04) :840-846
[9]
Inhibition of human mast cell proliferation and survival by tamoxifen in association with ion channel modulation [J].
Duffy, SM ;
Lawley, WJ ;
Kaur, D ;
Yang, WD ;
Bradding, P .
JOURNAL OF ALLERGY AND CLINICAL IMMUNOLOGY, 2003, 112 (05) :965-972
[10]
Segmental allergen challenge in patients with atopic asthma leads to increased IL-9 expression in bronchoalveolar lavage fluid lymphocytes [J].
Erpenbeck, VJ ;
Hohlfeld, JM ;
Volkmann, B ;
Hagenberg, A ;
Geldmacher, H ;
Braun, A ;
Krug, N .
JOURNAL OF ALLERGY AND CLINICAL IMMUNOLOGY, 2003, 111 (06) :1319-1327