The vitamin D receptor is required for mouse hair cycle progression but not for maintenance of the epidermal stem cell compartment

被引：22

作者：

Palmer, Hector G. ^{[1
]}

Martinez, Dolores

Carrneliet, Geert ^{[2
]}

Watt, Fiona M. ^{[1
,3
]}

机构：

[1] Li Ka Shing Ctr, Canc Res UK Cambridge Res Inst, Cambridge, England

[2] Katholieke Univ Leuven, Lab Expt Med & Endocrinol, Louvain, Belgium

[3] Univ Cambridge, Wellcome Trust Ctr Stem Cell Res, Cambridge, England

来源：

JOURNAL OF INVESTIGATIVE DERMATOLOGY | 2008年 / 128卷 / 08期

基金：

英国医学研究理事会;

关键词：

D O I：

10.1038/jid.2008.43

中图分类号：

R75 [皮肤病学与性病学];

学科分类号：

100206 ;

摘要：

引用

页码：2113 / 2117

页数：5

共 15 条

[1] Stem cell depletion through epidermal deletion of Rac1 [J].

Benitah, SA ;

Frye, M ;

Glogauer, M ;

Watt, FM .

SCIENCE, 2005, 309 (5736) :933-935

[2] Development and progression of alopecia in the vitamin D receptor null mouse [J].

Bikle, DD ;

Elalieh, H ;

Chang, S ;

Xie, ZJ ;

Sundberg, JP .

JOURNAL OF CELLULAR PHYSIOLOGY, 2006, 207 (02) :340-353

[3] Manipulation of stem cell proliferation and lineage commitment: visualisation of label-retaining cells in wholemounts of mouse epidermis [J].

Braun, KM ;

Niemann, C ;

Jensen, UB ;

Sundberg, JP ;

Silva-Vargas, V ;

Watt, FM .

DEVELOPMENT, 2003, 130 (21) :5241-5255

[4] Vitamin D receptor is essential for normal keratinocyte stem cell function [J].

Cianferotti, Luisella ;

Cox, Megan ;

Skorija, Kristi ;

Demay, Marie B. .

PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 2007, 104 (22) :9428-9433

[5] Vitamin D signalling pathways in cancer: potential for anticancer therapeutics [J].

Deeb, Kristin K. ;

Trump, Donald L. ;

Johnson, Candace S. .

NATURE REVIEWS CANCER, 2007, 7 (09) :684-700

[6]

Ghyselinck NB, 2002, ANN DERMATOL VENER, V129, P793

[7] Physical and functional interaction between the vitamin D receptor and hairless corepressor, two proteins required for hair cycling [J].

Hsieh, JC ;

Sisk, JM ;

Jurutka, PW ;

Haussler, CA ;

Slater, SA ;

Haussler, MR ;

Thompson, CC .

JOURNAL OF BIOLOGICAL CHEMISTRY, 2003, 278 (40) :38665-38674

[8] GENETIC-DEFECTS OF THE 1,25-DIHYDROXYVITAMIN-D3 RECEPTOR [J].

HUGHES, MR ;

MALLOY, PJ ;

OMALLEY, BW ;

PIKE, JW ;

FELDMAN, D .

JOURNAL OF RECEPTOR RESEARCH, 1991, 11 (1-4) :699-716

[9] MECHANISM OF SKIN MORPHOGENESIS .1. ANALYSES WITH ANTIBODIES TO ADHESION MOLECULES TENASCIN, N-CAM, AND INTEGRIN [J].

JIANG, TX ;

CHUONG, CM .

DEVELOPMENTAL BIOLOGY, 1992, 150 (01) :82-98

[10] Atrichia caused by mutations in the vitamin D receptor gene is a phenocopy of generalized atrichia caused by mutations in the hairless gene [J].

Miller, J ;

Djabali, K ;

Chen, T ;

Liu, Y ;

Ioffreda, M ;

Lyle, S ;

Christiano, AM ;

Holick, M ;

Cotsarelis, G .

JOURNAL OF INVESTIGATIVE DERMATOLOGY, 2001, 117 (03) :612-617

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