NO and cardiac diastolic function

被引：93

作者：

Paulus, WJ ^{[1
]}

Shah, AM

机构：

[1] Onze Lieve Vrouw Hosp, Ctr Cardiovasc, Aalst, Belgium

[2] Kings Coll London, Dept Cardiol, GKT Sch Med, London WC2R 2LS, England

来源：

CARDIOVASCULAR RESEARCH | 1999年 / 43卷 / 03期

关键词：

clinical; experimental; heart; general physiology; pathophysiology; nitric oxide; diastole; endothelial function; heart failure; ventricular function;

D O I：

10.1016/S0008-6363(99)00151-0

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

[No abstract available]

引用

页码：595 / 606

页数：12

共 92 条

[1] ENHANCEMENT OF LEFT-VENTRICULAR RELAXATION IN THE ISOLATED HEART BY AN ANGIOTENSIN-CONVERTING ENZYME-INHIBITOR [J].

ANNING, PB ;

GROCOTTMASON, RM ;

LEWIS, MJ ;

SHAH, AM .

CIRCULATION, 1995, 92 (09) :2660-2665

[2] Dobutamine enhances cardiodepressant effects of receptor-mediated coronary endothelial stimulation [J].

Bartunek, J ;

Shah, AM ;

Vanderheyden, M ;

Paulus, WJ .

CIRCULATION, 1997, 95 (01) :90-96

[3] Vasodilator dysfunction in aged spontaneously hypertensive rats:: changes in NO synthase III and soluble guanylyl cyclase expression, and in superoxide anion production [J].

Bauersachs, J ;

Bouloumié, A ;

Mülsch, A ;

Wiemer, G ;

Fleming, I ;

Busse, R .

CARDIOVASCULAR RESEARCH, 1998, 37 (03) :772-779

[4] Selective dysregulation of nitric oxide synthase type 3 in cardiac myocytes but not coronary microvascular endothelial cells of spontaneously hypertensive rat [J].

Bayraktutan, U ;

Yang, ZK ;

Shah, AM .

CARDIOVASCULAR RESEARCH, 1998, 38 (03) :719-726

[5] APPARENT HYDROXYL RADICAL PRODUCTION BY PEROXYNITRITE - IMPLICATIONS FOR ENDOTHELIAL INJURY FROM NITRIC-OXIDE AND SUPEROXIDE [J].

BECKMAN, JS ;

BECKMAN, TW ;

CHEN, J ;

MARSHALL, PA ;

FREEMAN, BA .

PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1990, 87 (04) :1620-1624

[6] EFFECTS OF SODIUM NITROPRUSSIDE ON LEFT-VENTRICULAR DIASTOLIC PRESSURE-VOLUME RELATIONS [J].

BRODIE, BR ;

GROSSMAN, W ;

MANN, T ;

MCLAURIN, LP .

JOURNAL OF CLINICAL INVESTIGATION, 1977, 59 (01) :59-68

[7] Nitric oxide, atrial natriuretic peptide, and cyclic GMP inhibit the growth-promoting effects of norepinephrine in cardiac myocytes and fibroblasts [J].

Calderone, A ;

Thaik, CM ;

Takahashi, N ;

Chang, DLF ;

Colucci, WS .

JOURNAL OF CLINICAL INVESTIGATION, 1998, 101 (04) :812-818

[8] Redox modulation of L-type calcium channels in ferret ventricular myocytes - Dual mechanism regulation by nitric oxide and S-nitrosothiols [J].

Campbell, DL ;

Stamler, JS ;

Strauss, HC .

JOURNAL OF GENERAL PHYSIOLOGY, 1996, 108 (04) :277-293

[9] The role of inducible nitric oxide synthase in cardiac allograft rejection [J].

Cannon, PJ ;

Yang, XC ;

Szabolcs, MJ ;

Ravalli, S ;

Sciacca, RR ;

Michler, RE .

CARDIOVASCULAR RESEARCH, 1998, 38 (01) :6-15

[10] THE DIFFERENTIAL-EFFECTS OF POSITIVE INOTROPIC AND VASODILATOR THERAPY ON DIASTOLIC PROPERTIES IN PATIENTS WITH CONGESTIVE CARDIOMYOPATHY [J].

CARROLL, JD ;

LANG, RM ;

NEUMANN, AL ;

BOROW, KM ;

RAJFER, SI .

CIRCULATION, 1986, 74 (04) :815-825

← 1 2 3 4 5 6 7 8 9 10 →