A new angle on a pervasive oncogene

[1] CATILLA LH, 1999, NAT GENET, V23, P144

[2] The murine gene p27Kip1 is haplo-insufficient for tumour suppression [J]. NATURE, 1998, 396 (6707) : 177 - 180

[3] Targeted disruption of Cbfa1 results in a complete lack of bone formation owing to maturational arrest of osteoblasts [J]. CELL, 1997, 89 (05) : 755 - 764

[4] FUSION BETWEEN TRANSCRIPTION FACTOR CBF-BETA/PEBP2-BETA AND A MYOSIN HEAVY-CHAIN IN ACUTE MYELOID-LEUKEMIA [J]. SCIENCE, 1993, 261 (5124) : 1041 - 1044

[5] Oncogenic transcription factors in the human acute leukemias [J]. SCIENCE, 1997, 278 (5340) : 1059 - 1064

[6] ETO, a target of t(8;21) in acute leukemia, interacts with the N-CoR and mSin3 corepressors [J]. MOLECULAR AND CELLULAR BIOLOGY, 1998, 18 (12) : 7176 - 7184

[7] Mutations involving the transcription factor CBFA1 cause cleidocranial dysplasia [J]. CELL, 1997, 89 (05) : 773 - 779

[8] AML1, the target of multiple chromosomal translocations in human leukemia, is essential for normal fetal liver hematopoiesis [J]. CELL, 1996, 84 (02) : 321 - 330

[9] Biallelic and heterozygous point mutations in the runt domain of the AML1/PEBP2αB gene associated with myeloblastic leukemias [J]. BLOOD, 1999, 93 (06) : 1817 - 1824

[10] Cbfa1, a candidate gene for cleidocranial dysplasia syndrome, is essential for osteoblast differentiation and bone development [J]. CELL, 1997, 89 (05) : 765 - 771

[1] CATILLA LH, 1999, NAT GENET, V23, P144

[2] The murine gene p27Kip1 is haplo-insufficient for tumour suppression [J]. NATURE, 1998, 396 (6707) : 177 - 180

[3] Targeted disruption of Cbfa1 results in a complete lack of bone formation owing to maturational arrest of osteoblasts [J]. CELL, 1997, 89 (05) : 755 - 764

[4] FUSION BETWEEN TRANSCRIPTION FACTOR CBF-BETA/PEBP2-BETA AND A MYOSIN HEAVY-CHAIN IN ACUTE MYELOID-LEUKEMIA [J]. SCIENCE, 1993, 261 (5124) : 1041 - 1044

[5] Oncogenic transcription factors in the human acute leukemias [J]. SCIENCE, 1997, 278 (5340) : 1059 - 1064

[6] ETO, a target of t(8;21) in acute leukemia, interacts with the N-CoR and mSin3 corepressors [J]. MOLECULAR AND CELLULAR BIOLOGY, 1998, 18 (12) : 7176 - 7184

[7] Mutations involving the transcription factor CBFA1 cause cleidocranial dysplasia [J]. CELL, 1997, 89 (05) : 773 - 779

[8] AML1, the target of multiple chromosomal translocations in human leukemia, is essential for normal fetal liver hematopoiesis [J]. CELL, 1996, 84 (02) : 321 - 330

[9] Biallelic and heterozygous point mutations in the runt domain of the AML1/PEBP2αB gene associated with myeloblastic leukemias [J]. BLOOD, 1999, 93 (06) : 1817 - 1824

[10] Cbfa1, a candidate gene for cleidocranial dysplasia syndrome, is essential for osteoblast differentiation and bone development [J]. CELL, 1997, 89 (05) : 765 - 771