Perspectives: Biomedicine - The benefits of recycling

被引:53
作者
Steel, KP [1 ]
机构
[1] MRC, Inst Hearing Res, Nottingham NG7 2RD, England
关键词
D O I
10.1126/science.285.5432.1363
中图分类号
O [数理科学和化学]; P [天文学、地球科学]; Q [生物科学]; N [自然科学总论];
学科分类号
07 ; 0710 ; 09 ;
摘要
引用
收藏
页码:1363 / 1364
页数:2
相关论文
共 12 条
[1]   Deafness and imbalance associated with inactivation of the secretory Ma-K-2Cl co-transporter [J].
Delpire, E ;
Lu, JM ;
England, R ;
Dull, C ;
Thorne, T .
NATURE GENETICS, 1999, 22 (02) :192-195
[2]   Mutation of the Na-K-Cl co-transporter gene Slc12a2 results in deafness in mice [J].
Dixon, MJ ;
Gazzard, J ;
Chaudhry, SS ;
Sampson, N ;
Schulte, BA ;
Steel, KP .
HUMAN MOLECULAR GENETICS, 1999, 8 (08) :1579-1584
[3]   Expression pattern of the mouse ortholog of the Pendred's syndrome gene (Pds) suggests a key role for pendrin in the inner ear [J].
Everett, LA ;
Morsli, H ;
Wu, DK ;
Green, ED .
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1999, 96 (17) :9727-9732
[4]   High prevalence of symptoms of Meniere's disease in three families with a mutation in the COCH gene [J].
Fransen, E ;
Verstreken, M ;
Verhagen, WIM ;
Wuyts, FL ;
Huygen, PLM ;
D'Haese, P ;
Robertson, NG ;
Morton, CC ;
McGuirt, WT ;
Smith, RJH ;
Declau, F ;
Van de Heyning, PH ;
Van Camp, G .
HUMAN MOLECULAR GENETICS, 1999, 8 (08) :1425-1429
[5]   Mutations in the gene encoding B1 subunit of H+-ATPase cause renal tubular acidosis with sensorineural deafness [J].
Karet, FE ;
Finberg, KE ;
Nelson, RD ;
Nayir, A ;
Mocan, H ;
Sanjad, SA ;
Rodriguez-Soriano, J ;
Santos, F ;
Cremers, CWRJ ;
Di Pietro, A ;
Hoffbrand, BI ;
Winiarski, J ;
Bakkaloglu, A ;
Ozen, S ;
Dusunsel, R ;
Goodyer, P ;
Hulton, SA ;
Wu, DK ;
Skvorak, AB ;
Morton, CC ;
Cunningham, MJ ;
Jha, V ;
Lifton, RP .
NATURE GENETICS, 1999, 21 (01) :84-90
[6]   GAP-JUNCTIONS IN THE RAT COCHLEA - IMMUNOHISTOCHEMICAL AND ULTRASTRUCTURAL ANALYSIS [J].
KIKUCHI, T ;
KIMURA, RS ;
PAUL, DL ;
ADAMS, JC .
ANATOMY AND EMBRYOLOGY, 1995, 191 (02) :101-118
[7]   KCNQ4, a novel potassium channel expressed in sensory outer hair cells, is mutated in dominant deafness [J].
Kubisch, C ;
Schroeder, BC ;
Friedrich, T ;
Lütjohann, B ;
El-Amraoui, A ;
Marlin, S ;
Petit, C ;
Jentsch, TJ .
CELL, 1999, 96 (03) :437-446
[8]   Altered cochlear fibrocytes in a mouse model of DFN3 nonsyndromic deafness [J].
Minowa, O ;
Ikeda, K ;
Sugitani, Y ;
Oshima, T ;
Nakai, S ;
Katori, Y ;
Suzuki, M ;
Furukawa, M ;
Kawase, T ;
Zheng, Y ;
Ogura, M ;
Asada, Y ;
Watanabe, K ;
Yamanaka, H ;
Gotoh, S ;
Nishi-Takeshima, M ;
Sugimoto, T ;
Kikuchi, T ;
Takasaka, T ;
Noda, T .
SCIENCE, 1999, 285 (5432) :1408-1411
[9]   Targeted mutagenesis of the POU-domain gene Brn4/Pou3f4 causes developmental defects in the inner ear [J].
Phippard, D ;
Lu, LH ;
Lee, D ;
Saunders, JC ;
Crenshaw, EB .
JOURNAL OF NEUROSCIENCE, 1999, 19 (14) :5980-5989
[10]   Deafness genes - expressions of surprise [J].
Steel, KP ;
Bussoli, TJ .
TRENDS IN GENETICS, 1999, 15 (06) :207-211