No link between the TFPI V264M mutation and venous thromboembolic disease

被引：22

作者：

Arnaud, E

Moatti, D

Emmerich, J

Aiach, M

de Prost, D

机构：

[1] CHU Xavier Bichat, Serv Hematol & Immunol, F-75018 Paris, France

[2] CHU Xavier Bichat, INSERM U479, F-75018 Paris, France

[3] CHU Broussais, INSERM U428, Paris, France

[4] CHU Broussais, Ctr Claude Bernard Malad Vasc, Paris, France

来源：

THROMBOSIS AND HAEMOSTASIS | 1999年 / 82卷 / 01期

关键词：

D O I：

10.1055/s-0037-1614654

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

引用

页码：159 / 160

页数：2

共 5 条

[1]

Alhenc-Gelas M, 1999, THROMB HAEMOSTASIS, V81, P506

[2]

Bajaj MS, 1997, THROMB HAEMOSTASIS, V78, P471

[3]

BROZE GJ, 1992, SEMIN HEMATOL, V29, P159

[4] Polymorphisms of the tissue factor pathway inhibitor (TFPI) gene in patients with acute coronary syndromes and in healthy subjects - Impact of the V264M substitution on plasma levels of TFPI [J].

Moatti, D ;

Seknadji, P ;

Galand, C ;

Poirier, O ;

Fumeron, F ;

Desprez, S ;

Garbarz, M ;

Dhermy, D ;

Arveiler, D ;

Evans, A ;

Luc, G ;

Ruidavets, JB ;

Ollivier, V ;

Hakim, J ;

Aumont, MC ;

de Prost, D .

ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY, 1999, 19 (04) :862-869

[5]

WUN TC, 1988, J BIOL CHEM, V263, P6001

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