Lack of BCL10 mutations in germ cell tumors and B cell lymphomas

[1] Baylin SB, 1998, ADV CANCER RES, V72, P141

[2] Molecular basis of hereditary persistence of fetal hemoglobin [J]. COOLEYS ANEMIA: SEVENTH SYMPOSIUM, 1998, 850 : 38 - 44

[3] Lee WC, 1996, CANCER RES, V56, P4297

[4] MATHEW S, 1994, CANCER RES, V54, P6265

[5] MURTY VVVS, 1994, ONCOGENE, V9, P2245

[6] OFFIT K, 1993, BLOOD, V82, P2157

[7] RAPID AND SENSITIVE DETECTION OF POINT MUTATIONS AND DNA POLYMORPHISMS USING THE POLYMERASE CHAIN-REACTION [J]. GENOMICS, 1989, 5 (04) : 874 - 879

[8] Bcl10 is involved in t(1;14)(p22;q32) of MALT B cell lymphoma and mutated in multiple tumor types [J]. CELL, 1999, 96 (01) : 35 - 45

[1] Baylin SB, 1998, ADV CANCER RES, V72, P141

[2] Molecular basis of hereditary persistence of fetal hemoglobin [J]. COOLEYS ANEMIA: SEVENTH SYMPOSIUM, 1998, 850 : 38 - 44

[3] Lee WC, 1996, CANCER RES, V56, P4297

[4] MATHEW S, 1994, CANCER RES, V54, P6265

[5] MURTY VVVS, 1994, ONCOGENE, V9, P2245

[6] OFFIT K, 1993, BLOOD, V82, P2157

[7] RAPID AND SENSITIVE DETECTION OF POINT MUTATIONS AND DNA POLYMORPHISMS USING THE POLYMERASE CHAIN-REACTION [J]. GENOMICS, 1989, 5 (04) : 874 - 879

[8] Bcl10 is involved in t(1;14)(p22;q32) of MALT B cell lymphoma and mutated in multiple tumor types [J]. CELL, 1999, 96 (01) : 35 - 45