Death receptors couple to both cell proliferation and apoptosis

[1] FAS ANTIGEN SIGNALS PROLIFERATION OF NORMAL HUMAN-DIPLOID FIBROBLAST AND ITS MECHANISM IS DIFFERENT FROM TUMOR-NECROSIS-FACTOR RECEPTOR [J]. FEBS LETTERS, 1995, 364 (01) : 5 - 8

[2] Early activation of caspases during T lymphocyte stimulation results in selective substrate cleavage in nonapoptotic cells [J]. JOURNAL OF EXPERIMENTAL MEDICINE, 1999, 190 (12) : 1879 - 1890

[3] CHANGES IN CELL CYCLE-ASSOCIATED GENE-EXPRESSION IN A MODEL OF IMPAIRED LIVER-REGENERATION [J]. FEBS LETTERS, 1994, 347 (2-3): : 157 - 162

[4] FAS TRANSDUCES ACTIVATION SIGNALS IN NORMAL HUMAN T-LYMPHOCYTES [J]. JOURNAL OF EXPERIMENTAL MEDICINE, 1993, 178 (06) : 2231 - 2235

[5] Human ICE/CED-3 protease nomenclature [J]. CELL, 1996, 87 (02) : 171 - 171

[6] Ashany D, 1999, J IMMUNOL, V163, P5303

[7] Fas receptor signaling inhibits glycogen synthase kinase 3β and induces cardiac hypertrophy following pressure overload [J]. JOURNAL OF CLINICAL INVESTIGATION, 2002, 109 (03) : 373 - 381

[8] UNRAVELING FUNCTION IN THE TNF LIGAND AND RECEPTOR FAMILIES [J]. SCIENCE, 1994, 264 (5159) : 667 - 668

[9] Development of inflammatory angiogenesis by local stimulation of Fas in vivo [J]. JOURNAL OF EXPERIMENTAL MEDICINE, 1997, 186 (01) : 147 - 152

[10] Proapoptotic Bcl-2 relative bim required for certain apoptotic responses, leukocyte homeostasis, and to preclude autoimmunity [J]. SCIENCE, 1999, 286 (5445) : 1735 - 1738

[1] FAS ANTIGEN SIGNALS PROLIFERATION OF NORMAL HUMAN-DIPLOID FIBROBLAST AND ITS MECHANISM IS DIFFERENT FROM TUMOR-NECROSIS-FACTOR RECEPTOR [J]. FEBS LETTERS, 1995, 364 (01) : 5 - 8

[2] Early activation of caspases during T lymphocyte stimulation results in selective substrate cleavage in nonapoptotic cells [J]. JOURNAL OF EXPERIMENTAL MEDICINE, 1999, 190 (12) : 1879 - 1890

[3] CHANGES IN CELL CYCLE-ASSOCIATED GENE-EXPRESSION IN A MODEL OF IMPAIRED LIVER-REGENERATION [J]. FEBS LETTERS, 1994, 347 (2-3): : 157 - 162

[4] FAS TRANSDUCES ACTIVATION SIGNALS IN NORMAL HUMAN T-LYMPHOCYTES [J]. JOURNAL OF EXPERIMENTAL MEDICINE, 1993, 178 (06) : 2231 - 2235

[5] Human ICE/CED-3 protease nomenclature [J]. CELL, 1996, 87 (02) : 171 - 171

[6] Ashany D, 1999, J IMMUNOL, V163, P5303

[7] Fas receptor signaling inhibits glycogen synthase kinase 3β and induces cardiac hypertrophy following pressure overload [J]. JOURNAL OF CLINICAL INVESTIGATION, 2002, 109 (03) : 373 - 381

[8] UNRAVELING FUNCTION IN THE TNF LIGAND AND RECEPTOR FAMILIES [J]. SCIENCE, 1994, 264 (5159) : 667 - 668

[9] Development of inflammatory angiogenesis by local stimulation of Fas in vivo [J]. JOURNAL OF EXPERIMENTAL MEDICINE, 1997, 186 (01) : 147 - 152

[10] Proapoptotic Bcl-2 relative bim required for certain apoptotic responses, leukocyte homeostasis, and to preclude autoimmunity [J]. SCIENCE, 1999, 286 (5445) : 1735 - 1738