Divergence of hedgehog signal transduction mechanism between Drosophila and mammals

被引:196
作者
Varjosalo, M
Li, SP
Taipale, J
机构
[1] Univ Helsinki, Biomedicum, Mol & Canc Biol Program, FIN-00014 Helsinki, Finland
[2] Natl Publ Hlth Inst, Dept Mol Med, FIN-00014 Helsinki, Finland
基金
芬兰科学院;
关键词
D O I
10.1016/j.devcel.2005.12.014
中图分类号
Q2 [细胞生物学];
学科分类号
071009 ; 090102 ;
摘要
The Hedgehog (Hh) signaling pathway has conserved roles in development of species ranging from Drosophila to humans. Responses to Hh are mediated by the transcription factor Cubitus interruptus (Ci; GLIs 1-3 in mammals), and constitutive activation of Hh target gene expression has been linked to several types of human cancer. In Drosophila, the kinesin-like protein Costal2 (Cos2), which associates directly with the Hh receptor component Smoothened (Smo), is essential for suppression of the transcriptional activity of Ci in the absence of ligand. Another protein, Suppressor of Fused (Su(Fu)), exerts a weak negative influence on Ci activity. Based on analysis of functional and sequence conservation of Cos2 orthologs, Su(Fu), Smo, and Ci/GLI proteins, we find here that Drosophila and mammalian Hh signaling mechanisms have diverged, and that, in mouse cells, major Cos2-like activities are absent and the inhibition of the Hh pathway in the absence of ligand critically depends on Su(Fu).
引用
收藏
页码:177 / 186
页数:10
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