Immunopathogenesis of crescentic glomerulonephritis

被引：28

作者：

Tipping, PG ^{[1
]}

Kitching, AR ^{[1
]}

Cunningham, MA ^{[1
]}

Holdsworth, SR ^{[1
]}

机构：

[1] Monash Univ, Monash Med Ctr, Dept Med, Ctr Inflammatory Dis, Clayton, Vic 3168, Australia

来源：

CURRENT OPINION IN NEPHROLOGY AND HYPERTENSION | 1999年 / 8卷 / 03期

关键词：

D O I：

10.1097/00041552-199905000-00002

中图分类号：

R5 [内科学]; R69 [泌尿科学（泌尿生殖系疾病）];

学科分类号：

1002 ; 100201 ;

摘要：

Crescentic glomerulonephritis provides an important therapeutic challenge because of its rapidly progressive course and poor outcome. Studies in animal models have elucidated some of the pivotal pathogenetic mechanisms, and human studies increasingly support the clinical relevance of these animal data. Accumulating evidence suggests that crescentic glomerulonephritis results from a complex cell-mediated nephritogenic immune response. Interruption of a number of immune and inflammatory mediators can improve the outcome of this disease. Curr Opin Nephrol Hypertens 8:281-286. (C) 1999 Lippincott Williams & Wilkins.

引用

页码：281 / 286

页数：6

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