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Role of ischemia-reperfusion injury in the development of chronic renal allograft damage

被引：41
作者：
Grinyo, JM [1 ]
机构：
[1] Univ Barcelona, Bellvitge Hosp, Serv Nefrol, Barcelona 08907, Spain
来源：
TRANSPLANTATION PROCEEDINGS | 2001年 / 33卷 / 7-8期
关键词：
D O I：
10.1016/S0041-1345(01)02527-1
中图分类号：
R392 [医学免疫学]; Q939.91 [免疫学];
学科分类号：
100102 ;
摘要：
[No abstract available]
引用
收藏
页码：3741 / 3742
页数：2
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    [J]. KIDNEY INTERNATIONAL, 2000, 58 (02) : 859 - 866
  • [2] Influence of nephron mass in development of chronic renal failure after prolonged warm renal ischemia
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    [J]. AMERICAN JOURNAL OF PHYSIOLOGY-RENAL PHYSIOLOGY, 2000, 279 (02) : F259 - F269
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    [J]. KIDNEY INTERNATIONAL, 2000, 58 (02) : 838 - 850
  • [5] Cold ischemia in the absence of alloreactivity induces chronic transplant nephropathy through a process mediated by the platelet-activating factor
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  • [6] Cold ischemic injury accelerates the progression to chronic rejection in a rat cardiac allograft model
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  • [7] Late graft dysfunction after prolonged cold ischemia of the donor kidney - Inhibition by cyclosporine
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    [J]. TRANSPLANTATION, 1999, 68 (07) : 1004 - 1010
  • [8] Exploitation of the continuum between early ischemia reperfusion injury and host alloresponsiveness - Indefinite kidney allograft survival by treatment with a soluble P-selectin ligand and low-dose cyclosporine in combination
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  • [10] Long-term graft outcome is not necessarily affected by delayed onset of graft function and early acute rejection
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    [J]. TRANSPLANTATION, 1997, 64 (01) : 103 - 107
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