Adiponectin and osteocalcin: relation to insulin sensitivity

被引:3
作者
Zhang, Yanjun [2 ]
Zhou, Peng [2 ]
Kimondo, Julia Wanjiru [1 ]
机构
[1] Tianjin Univ Tradit Chinese Med, Dept Pharmacol, Tianjin 30086, Peoples R China
[2] Tianjin Univ Tradit Chinese Med, Sch Chinese Med, Tianjin 30086, Peoples R China
关键词
adiponectin; osteocalcin; differentiation; PPAR; BMP-2; MARROW STROMAL CELLS; BONE-MINERAL DENSITY; PPAR-GAMMA; OSTEOBLAST DIFFERENTIATION; TRANSCRIPTION FACTORS; METABOLIC SYNDROME; PREADIPOCYTE DIFFERENTIATION; NONHEMATOPOIETIC TISSUES; SKELETAL DEVELOPMENT; DIABETES-MELLITUS;
D O I
10.1139/O2012-022
中图分类号
Q5 [生物化学]; Q7 [分子生物学];
学科分类号
070307 [化学生物学]; 071010 [生物化学与分子生物学];
摘要
Obesity and osteoporosis have grave consequences for human health, quality of life, and even the efficiency of the labor force. Interestingly, these diseases share several features including a genetic predisposition and a common progenitor cell. Recent findings show that high adipocyte count in bone marrow is directly related to bone loss, as fat cells replace osteoblasts resulting in reduced bone mineral density and increased propensity towards osteoporosis. This close relationship has a positive aspect, whereby higher osteocalcin levels results in increased adiponectin production while the presence of adiponectin influences osteoblast proliferation and differentiation in a positive way. We focus on how osteoblasts and adipocytes affect each other and ultimately insulin resistance through the hormones they produce. This approach to whole animal physiology is the main stay of Alternative Medicine. It is assumed that the body is linked together intricately, and treating one is equal to treating the whole body. As we go further into bone and adipocytes physiology, it is evident that these organs affect each other. Therefore, elucidation on the actions of fat on bone and vice versa will unravel the complex mechanism of insulin resistance.
引用
收藏
页码:613 / 620
页数:8
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