Health inequalities and the psychosocial environment

被引:17
作者
Marmot, M [1 ]
Siegrist, J [1 ]
机构
[1] Univ Dusseldorf, Dept Med Sociol, D-40225 Dusseldorf, Germany
基金
英国医学研究理事会;
关键词
Effort-reward imbalance; Europe; Job strain; Psychosocial environment; Social inequalities and health; Stressful work;
D O I
10.1016/S0277-9536(03)00348-4
中图分类号
R1 [预防医学、卫生学];
学科分类号
1004 ; 120402 ;
摘要
As social inequalities in health continue to be a key public health problem, scientific advances in explaining these inequalities are needed. It is unlikely that there will be a single explanation of social inequalities in health. This introductory paper sets out one explanatory framework, exposure to adverse psychosocial environments during midlife, and particularly at work. We argue that exposure to an adverse psychosocial environment, in terms of job tasks, defined by high demands and low control and/or by effort-reward imbalance, elicits sustained stress reactions with negative long-term consequences for health. These exposures may be implicated in the association of socioeconomic status with health in two ways. First, these exposures are likely to be experienced more frequently among lower socioeconomic groups. Second, the size of the effects on health produced by adverse working conditions may be higher in lower status groups, due to their increased vulnerability. In this special issue, these arguments are illustrated by a collection of original contributions from collaborative research across Europe. The papers, in our view, advance the case for the robust associations between measures of adverse psychosocial environment and ill health, as they are based on comparative studies across several European countries and as they combine different types of study designs. This collaboration was enabled and supported by a European Science Foundation scientific programme on 'Social Variations in Health Expectancy in Europe'. © 2003 Published by Elsevier Ltd.
引用
收藏
页码:1461 / 1461
页数:1
相关论文
共 73 条
[1]  
[Anonymous], HLTH INEQUALITIES
[2]  
[Anonymous], 1989, Health inequalities in European countries
[3]  
[Anonymous], 1998, THEORIES ORG STRESS
[4]  
Bandura A., 1996, SOCIAL FDN THOUGHT A
[5]   FETAL ORIGINS OF CORONARY HEART-DISEASE [J].
BARKER, DJP .
BRITISH MEDICAL JOURNAL, 1995, 311 (6998) :171-174
[6]  
Berkman L., 2000, Social Epidemiology, P137, DOI DOI 10.1016/S0277-9536(00)00065-4
[7]   A mechanism converting psychosocial stress into mononuclear cell activation [J].
Bierhaus, A ;
Wolf, J ;
Andrassy, M ;
Rohleder, N ;
Humpert, PM ;
Petrov, D ;
Ferstl, R ;
von Eynatten, M ;
Wendt, T ;
Rudofsky, G ;
Joswig, M ;
Morcos, M ;
Schwaninger, M ;
McEwen, B ;
Kirschbaum, C ;
Nawroth, PP .
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 2003, 100 (04) :1920-1925
[8]  
Blum K, 1996, AM SCI, V84, P132
[9]   Association between psychosocial factors at work and nonfatal myocardial infarction in a population-based case-control study in Czech men [J].
Bobák, M ;
Hertzman, C ;
Skodová, Z ;
Marmot, M .
EPIDEMIOLOGY, 1998, 9 (01) :43-47
[10]   Two alternative job stress models and the risk of coronary heart disease [J].
Bosma, H ;
Peter, R ;
Siegrist, J ;
Marmot, M .
AMERICAN JOURNAL OF PUBLIC HEALTH, 1998, 88 (01) :68-74