Chemokines active on eosinophils: Potential roles in allergic inflammation

[1] Increased MCP-1, RANTES, and MIP-1 alpha in bronchoalveolar lavage fluid of allergic asthmatic patients [J]. AMERICAN JOURNAL OF RESPIRATORY AND CRITICAL CARE MEDICINE, 1996, 153 (04) : 1398 - 1404

[2] ACTIVATION OF DUAL T-CELL SIGNALING PATHWAYS BY THE CHEMOKINE RANTES [J]. SCIENCE, 1995, 269 (5231) : 1727 - 1730

[3] BECH LA, 1994, J ALLERGY CLIN IMMUN, V93, P234

[4] EOSINOPHILIC INFLAMMATION IN ASTHMA [J]. NEW ENGLAND JOURNAL OF MEDICINE, 1990, 323 (15) : 1033 - 1039

[5] EOSINOPHILS, T-LYMPHOCYTES, MAST-CELLS, NEUTROPHILS, AND MACROPHAGES IN BRONCHIAL BIOPSY SPECIMENS FROM ATOPIC SUBJECTS WITH ASTHMA - COMPARISON WITH BIOPSY SPECIMENS FROM ATOPIC SUBJECTS WITHOUT ASTHMA AND NORMAL CONTROL SUBJECTS AND RELATIONSHIP TO BRONCHIAL HYPERRESPONSIVENESS [J]. JOURNAL OF ALLERGY AND CLINICAL IMMUNOLOGY, 1991, 88 (04) : 661 - 674

[6] BUTTERFIELD JH, 1994, SAMTERS IMMUNOLOGIC, P501

[7] MOLECULAR-CLONING AND FUNCTIONAL EXPRESSION OF 2 MONOCYTE CHEMOATTRACTANT PROTEIN-1 RECEPTORS REVEALS ALTERNATIVE SPLICING OF THE CARBOXYL-TERMINAL TAILS [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1994, 91 (07) : 2752 - 2756

[8] IMMUNOGLOBULIN E-MEDIATED INCREASE IN VASCULAR-PERMEABILITY CORRELATES WITH EOSINOPHILIC INFLAMMATION [J]. AMERICAN REVIEW OF RESPIRATORY DISEASE, 1993, 147 (03): : 677 - 683

[9] COOPERATION BETWEEN INTERLEUKIN-5 AND THE CHEMOKINE EOTAXIN TO INDUCE EOSINOPHIL ACCUMULATION IN-VIVO [J]. JOURNAL OF EXPERIMENTAL MEDICINE, 1995, 182 (04) : 1169 - 1174

[10] MONOCYTE CHEMOTACTIC PROTEIN-3 IS A MOST EFFECTIVE BASOPHIL-ACTIVATING AND EOSINOPHIL-ACTIVATING CHEMOKINE [J]. JOURNAL OF EXPERIMENTAL MEDICINE, 1994, 179 (02) : 751 - 756

[1] Increased MCP-1, RANTES, and MIP-1 alpha in bronchoalveolar lavage fluid of allergic asthmatic patients [J]. AMERICAN JOURNAL OF RESPIRATORY AND CRITICAL CARE MEDICINE, 1996, 153 (04) : 1398 - 1404

[2] ACTIVATION OF DUAL T-CELL SIGNALING PATHWAYS BY THE CHEMOKINE RANTES [J]. SCIENCE, 1995, 269 (5231) : 1727 - 1730

[3] BECH LA, 1994, J ALLERGY CLIN IMMUN, V93, P234

[4] EOSINOPHILIC INFLAMMATION IN ASTHMA [J]. NEW ENGLAND JOURNAL OF MEDICINE, 1990, 323 (15) : 1033 - 1039

[5] EOSINOPHILS, T-LYMPHOCYTES, MAST-CELLS, NEUTROPHILS, AND MACROPHAGES IN BRONCHIAL BIOPSY SPECIMENS FROM ATOPIC SUBJECTS WITH ASTHMA - COMPARISON WITH BIOPSY SPECIMENS FROM ATOPIC SUBJECTS WITHOUT ASTHMA AND NORMAL CONTROL SUBJECTS AND RELATIONSHIP TO BRONCHIAL HYPERRESPONSIVENESS [J]. JOURNAL OF ALLERGY AND CLINICAL IMMUNOLOGY, 1991, 88 (04) : 661 - 674

[6] BUTTERFIELD JH, 1994, SAMTERS IMMUNOLOGIC, P501

[7] MOLECULAR-CLONING AND FUNCTIONAL EXPRESSION OF 2 MONOCYTE CHEMOATTRACTANT PROTEIN-1 RECEPTORS REVEALS ALTERNATIVE SPLICING OF THE CARBOXYL-TERMINAL TAILS [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1994, 91 (07) : 2752 - 2756

[8] IMMUNOGLOBULIN E-MEDIATED INCREASE IN VASCULAR-PERMEABILITY CORRELATES WITH EOSINOPHILIC INFLAMMATION [J]. AMERICAN REVIEW OF RESPIRATORY DISEASE, 1993, 147 (03): : 677 - 683

[9] COOPERATION BETWEEN INTERLEUKIN-5 AND THE CHEMOKINE EOTAXIN TO INDUCE EOSINOPHIL ACCUMULATION IN-VIVO [J]. JOURNAL OF EXPERIMENTAL MEDICINE, 1995, 182 (04) : 1169 - 1174

[10] MONOCYTE CHEMOTACTIC PROTEIN-3 IS A MOST EFFECTIVE BASOPHIL-ACTIVATING AND EOSINOPHIL-ACTIVATING CHEMOKINE [J]. JOURNAL OF EXPERIMENTAL MEDICINE, 1994, 179 (02) : 751 - 756