Differing influences of virus burden and immune activation on disease severity in secondary dengue-3 virus infections

被引:383
作者
Libraty, DH
Endy, TP
Houng, HSH
Green, S
Kalayanarooj, S
Suntayakorn, S
Chansiriwongs, W
Vaughn, DW
Nisalak, A
Ennis, FA
Rothman, AL
机构
[1] Univ Massachusetts, Sch Med, Ctr Infect Dis & Vaccine Res, Worcester, MA 01655 USA
[2] Walter Reed Army Inst Res, Dept Virus Dis, Silver Spring, MD USA
[3] Queen Sirikit Natl Inst Child Hlth, Dept Pediat, Bangkok, Thailand
[4] Kamphaeng Phet Prov Hosp, Dept Pediat, Kamphaeng Phet, Thailand
关键词
D O I
10.1086/340365
中图分类号
R392 [医学免疫学]; Q939.91 [免疫学];
学科分类号
100102 ;
摘要
Dengue hemorrhagic fever (DHF), the most severe form of illness following infection with a dengue virus, is characterized by plasma leakage, thrombocytopenia, and hepatic inflammation. The interrelationships among virus burden, immune activation, and development of DHF were examined in 54 children with secondary dengue-3 virus infections participating in a prospective, hospital-based study. DHF was associated with higher mean plasma viremia early in illness and earlier peak plasma interferon-gamma levels. Maximum plasma viremia levels correlated with the degree of plasma leakage and thrombocytopenia. Maximum plasma levels of interleukin (IL)-10 and soluble tumor necrosis factor receptor-II correlated with the degree of thrombocytopenia, independently of viremia levels. Hepatic transaminase elevation correlated with plasma soluble IL-2 receptor levels and not with viremia levels. Quantitative differences in virus burden and host immune responses, and the timing of type 1 cytokine responses, have differing influences on the severity of disease manifestations during secondary dengue-3 virus infections.
引用
收藏
页码:1213 / 1221
页数:9
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