Enhancement of cellular adenosine triphosphate levels in PC12 cells by extracellular adenosine

被引:10
作者
Fujimori, H [1 ]
Yasuda, M [1 ]
Pan-Hou, H [1 ]
机构
[1] Setsunan Univ, Fac Pharmaceut Sci, Osaka 5730101, Japan
关键词
PC12; cell; adenosine; ATP synthesis; salvage pathway; P1; receptor;
D O I
10.1248/bpb.25.307
中图分类号
R9 [药学];
学科分类号
1007 ;
摘要
To elucidate the biological significance of extracellular adenine compounds, the effects of adenosine (Ado) on cellular levels of adenine compounds, especially adenosine triphosphate (ATP), in PC12 cells were studied. Ado and inosine but not adenosine 5'-monophosphate, adenosine 5'-diphosphate, ATP, guanosine, cytosine, thymidine, and uridine, significantly enhanced cellular ATP levels in PC12 cells in time- and dose-dependent manners. Various P1 receptor agonists of Ado did not enhance the ATP level. In addition, theophylline, an antagonist of P1 receptors, did not inhibit the Ado-evoked ATP enhancement. These results suggest that the Ado receptor is not involved in the augmentation of the cellular ATP level induced by Ado in PC12 cells. The ATP-enhancing effect of Ado was potentiated by dipyridamole, an inhibitor of Ado uptake, or coformycin, an inhibitor of Ado deaminase. The effect of Ado on the ATP level was also observed when PC12 cells were incubated in glucose-free medium. Together these results suggest that enhancement of cellular ATP levels in PC12 cells by extracellular Ado might be acceleration of ATP synthesis through the Ado salvage system using hypoxanthine-guanine phosphoribosyltransferase rather than Ado kinase since 5'-iodotubercidin, an inhibitor of Ado kinase, had no effect on the enhancement elicited by Ado.
引用
收藏
页码:307 / 311
页数:5
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