Interaction of bradykinin with angiotensin, prostacyclin, and nitric oxide in myocardial preservation

被引：4

作者：

Sato, M

Das, DK ^{[1
]}

Engelman, RM

机构：

[1] Univ Connecticut, Sch Med, Dept Surg, Farmington, CT 06030 USA

[2] Baystate Med Ctr, Dept Cardiac Surg, Springfield, MA 01199 USA

来源：

HEART IN STRESS | 1999年 / 874卷

关键词：

D O I：

10.1111/j.1749-6632.1999.tb09244.x

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

This review focuses on the importance of bradykinin in myocardial preservation during ischemic arrest. Bradykinin is released from the heart spontaneously in response to ischemic stress, which may be viewed as a survival signal of the heart against Ischemia, Bradykinin appears to function as a signaling molecule by controlling the release of other intracellular modulators, such as prostacyclins and nitric oxide, which also exert beneficial effects on the ischemic myocardium.

引用

收藏

页码：286 / 294

页数：9

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