Oxidative stress - The curse that underlies cerebral vascular dysfunction?

被引:59
作者
Faraci, FM
机构
[1] Univ Iowa, Dept Internal Med, Carver Coll Med, Ctr Cardiovasc, Iowa City, IA 52242 USA
[2] Univ Iowa, Dept Pharmacol, Carver Coll Med, Ctr Cardiovasc, Iowa City, IA 52242 USA
关键词
D O I
10.1161/01.STR.0000153067.27288.8b
中图分类号
R74 [神经病学与精神病学];
学科分类号
摘要
[No abstract available]
引用
收藏
页码:186 / 188
页数:3
相关论文
共 34 条
[1]   Structure of cerebral arterioles in mice deficient in expression of the gene for endothelial nitric oxide synthase [J].
Baumbach, GL ;
Sigmund, CD ;
Faraci, FM .
CIRCULATION RESEARCH, 2004, 95 (08) :822-829
[2]  
Baumbach GL, 2003, FASEB J, V17, pA63
[3]   Evidence that Rho-kinase activity contributes to cerebral vascular tone in vivo and is enhanced during chronic hypertension - Comparison with protein kinase C [J].
Chrissobolis, S ;
Sobey, CG .
CIRCULATION RESEARCH, 2001, 88 (08) :774-779
[4]   Cerebral vascular dysfunction mediated by superoxide in hyperhomocysteinemic mice [J].
Dayal, S ;
Arning, E ;
Bottiglieri, T ;
Böger, RH ;
Sigmund, CD ;
Faraci, FM ;
Lentz, SR .
STROKE, 2004, 35 (08) :1957-1962
[5]   Impaired endothelium-dependent responses and enhanced influence of Rho-kinase in cerebral arterioles in type II diabetes [J].
Didion, SP ;
Lynch, CM ;
Baumbach, GL ;
Faraci, FM .
STROKE, 2005, 36 (02) :342-347
[6]   Increased superoxide and vascular dysfunction in CuZnSOD-deficient mice [J].
Didion, SP ;
Ryan, MJ ;
Didion, LA ;
Fegan, PE ;
Sigmund, CD ;
Faraci, FM .
CIRCULATION RESEARCH, 2002, 91 (10) :938-944
[7]  
DIDION SP, IN PRESS FASEB J
[8]   Free radicals in the physiological control of cell function [J].
Dröge, W .
PHYSIOLOGICAL REVIEWS, 2002, 82 (01) :47-95
[9]   Inhibitory effect of recombinant iNOS gene expression on vasomotor function of canine basilar artery [J].
Eguchi, D ;
D'Uscio, LV ;
Wambi, C ;
Weiler, D ;
Kovesdi, I ;
O'Brien, T ;
Katusic, ZS .
AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY, 2002, 283 (06) :H2560-H2566
[10]   Cerebrovascular dysfunction in Zucker obese rats is mediated by oxidative stress and protein kinase C [J].
Erdös, B ;
Snipes, JA ;
Miller, AW ;
Busija, DW .
DIABETES, 2004, 53 (05) :1352-1359