Mechanotransduction in cardiomyocyte hypertrophy

[1] Tyrosine kinase and c-Jun NH2-terminal kinase mediate hypertrophic responses to prostaglandin F2α in cultured neonatal rat ventricular myocytes [J]. CIRCULATION RESEARCH, 1998, 83 (02) : 167 - 178

[2] Oxidative stress activates extracellular signal-regulated kinases through Src and ras in cultured cardiac myocytes of neonatal rats [J]. JOURNAL OF CLINICAL INVESTIGATION, 1997, 100 (07) : 1813 - 1821

[3] The MEK1-ERK1/2 signaling pathway promotes compensated cardiac hypertrophy in transgenic mice [J]. EMBO JOURNAL, 2000, 19 (23) : 6341 - 6350

[4] PRESSURE-INDUCED AND VOLUME-INDUCED LEFT-VENTRICULAR HYPERTROPHIES ARE ASSOCIATED WITH DISTINCT MYOCYTE PHENOTYPES AND DIFFERENTIAL INDUCTION OF PEPTIDE GROWTH-FACTOR MESSENGER-RNAS [J]. CIRCULATION, 1995, 92 (09) : 2385 - 2390

[5] Stimulation of multiple mitogen-activated protein kinase sub-families by oxidative stress and phosphorylation of the small heat shock protein, HSP25/27, in neonatal ventricular myocytes [J]. BIOCHEMICAL JOURNAL, 1998, 333 : 581 - 589

[6] The search for physiological substrates of MAP and SAP kinases in mammalian cells [J]. TRENDS IN CELL BIOLOGY, 1997, 7 (09) : 353 - 361

[7] Cook SA, 1999, CIRC RES, V85, P940

[8] Regional myosin heavy chain expression in volume and pressure overload induced cardiac hypertrophy [J]. ACTA PHYSIOLOGICA SCANDINAVICA, 1995, 155 (04): : 397 - 404

[9] Stretch-activated ion channels in the heart [J]. JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY, 1997, 29 (06) : 1511 - 1523

[10] Mechanical strain activates BNP gene transcription through a p38/NF-κB-dependent mechanism [J]. JOURNAL OF CLINICAL INVESTIGATION, 1999, 104 (11) : 1603 - 1612

[1] Tyrosine kinase and c-Jun NH2-terminal kinase mediate hypertrophic responses to prostaglandin F2α in cultured neonatal rat ventricular myocytes [J]. CIRCULATION RESEARCH, 1998, 83 (02) : 167 - 178

[2] Oxidative stress activates extracellular signal-regulated kinases through Src and ras in cultured cardiac myocytes of neonatal rats [J]. JOURNAL OF CLINICAL INVESTIGATION, 1997, 100 (07) : 1813 - 1821

[3] The MEK1-ERK1/2 signaling pathway promotes compensated cardiac hypertrophy in transgenic mice [J]. EMBO JOURNAL, 2000, 19 (23) : 6341 - 6350

[4] PRESSURE-INDUCED AND VOLUME-INDUCED LEFT-VENTRICULAR HYPERTROPHIES ARE ASSOCIATED WITH DISTINCT MYOCYTE PHENOTYPES AND DIFFERENTIAL INDUCTION OF PEPTIDE GROWTH-FACTOR MESSENGER-RNAS [J]. CIRCULATION, 1995, 92 (09) : 2385 - 2390

[5] Stimulation of multiple mitogen-activated protein kinase sub-families by oxidative stress and phosphorylation of the small heat shock protein, HSP25/27, in neonatal ventricular myocytes [J]. BIOCHEMICAL JOURNAL, 1998, 333 : 581 - 589

[6] The search for physiological substrates of MAP and SAP kinases in mammalian cells [J]. TRENDS IN CELL BIOLOGY, 1997, 7 (09) : 353 - 361

[7] Cook SA, 1999, CIRC RES, V85, P940

[8] Regional myosin heavy chain expression in volume and pressure overload induced cardiac hypertrophy [J]. ACTA PHYSIOLOGICA SCANDINAVICA, 1995, 155 (04): : 397 - 404

[9] Stretch-activated ion channels in the heart [J]. JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY, 1997, 29 (06) : 1511 - 1523

[10] Mechanical strain activates BNP gene transcription through a p38/NF-κB-dependent mechanism [J]. JOURNAL OF CLINICAL INVESTIGATION, 1999, 104 (11) : 1603 - 1612