Use of an anti-interleukin-5 antibody in the hypereosinophilic syndrome with eosinophilic dermatitis

被引：199

作者：

Plotz, S

Simon, H

Darsow, U

Simon, D

Vassina, E

Yousefi, S

Hein, R

Smith, T

Behrendt, H

Ring, J

机构：

[1] Univ Bern, Dept Pharmacol, CH-3010 Bern, Switzerland

[2] Tech Univ Munich, Div Environm Dermatol & Allergy GSF, TUM, Ctr Allergy & Environm, D-8000 Munich, Germany

[3] Tech Univ Munich, Dept Dermatol & Allergy, D-8000 Munich, Germany

[4] Univ Bern, Dept Dermatol, Bern, Switzerland

[5] GlaxoSmithKline, Harlow, Essex, England

来源：

NEW ENGLAND JOURNAL OF MEDICINE | 2003年 / 349卷 / 24期

关键词：

D O I：

10.1056/NEJMoa031261

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

[No abstract available]

引用

页码：2334 / 2339

页数：6

共 16 条

[1] HYPEREOSINOPHILIC SYNDROME - ANALYSIS OF 14 CASES WITH REVIEW OF LITERATURE [J].

CHUSID, MJ ;

DALE, DC ;

WEST, BC ;

WOLFF, SM .

MEDICINE, 1975, 54 (01) :1-27

[2] BRIEF REPORT - CLONAL PROLIFERATION OF TYPE-2 HELPER T-CELLS IN A MAN WITH THE HYPEREOSINOPHILIC SYNDROME [J].

COGAN, E ;

SCHANDENE, L ;

CRUSIAUX, A ;

COCHAUX, P ;

VELU, T ;

GOLDMAN, M .

NEW ENGLAND JOURNAL OF MEDICINE, 1994, 330 (08) :535-538

[3] A tyrosine kinase created by fusion of the PDGFRA and FIP1L1 genes as a therapeutic target of imatinib in idiopathic hypereosinophilic syndrome [J].

Cools, J ;

DeAngelo, DJ ;

Gotlib, J ;

Stover, EH ;

Legare, RD ;

Cortes, J ;

Kutok, J ;

Clark, J ;

Galinsky, I ;

Griffin, JD ;

Cross, NCP ;

Tefferi, A ;

Malone, J ;

Alam, R ;

Schrier, SL ;

Schmid, J ;

Rose, M ;

Vandenberghe, P ;

Verhoef, G ;

Boogaerts, M ;

Wlodarska, I ;

Kantarjian, H ;

Marynen, P ;

Coutre, SE ;

Stone, R ;

Gilliland, DG .

NEW ENGLAND JOURNAL OF MEDICINE, 2003, 348 (13) :1201-1214

[4] High serum thymus and activation-regulated chemokine levels in the lymphocytic variant of the hypereosinophilic syndrome [J].

de Lavareille, A ;

Roufosse, F ;

Schmid-Grendelmeier, P ;

Roumier, AS ;

Schandené, L ;

Cogan, E ;

Simon, HU ;

Goldman, M .

JOURNAL OF ALLERGY AND CLINICAL IMMUNOLOGY, 2002, 110 (03) :476-479

[5] Eosinophil's role remains uncertain as anti-interleukin-5 only partially depletes numbers in asthmatic airway [J].

Flood-Page, PT ;

Menzies-Gow, AN ;

Kay, AB ;

Robinson, DS .

AMERICAN JOURNAL OF RESPIRATORY AND CRITICAL CARE MEDICINE, 2003, 167 (02) :199-204

[6] Treatment of hypereosinophilic syndrome with imatinib mesilate [J].

Gleich, GJ ;

Leiferman, KM ;

Pardanani, A ;

Tefferi, A ;

Butterfield, JH .

LANCET, 2002, 359 (9317) :1577-1578

[7] Discovery of a fusion kinase in EOL-1 cells and idiopathic hypereosinophilic syndrome [J].

Griffin, JH ;

Leung, J ;

Bruner, RJ ;

Caligiuri, MA ;

Briesewitz, R .

PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 2003, 100 (13) :7830-7835

[8] DERMATOLOGIC MANIFESTATIONS OF HYPEREOSINOPHILIC SYNDROME [J].

KAZMIEROWSKI, JA ;

CHUSID, MJ ;

PARRILLO, JE ;

FAUCI, AS ;

WOLFF, SM .

ARCHIVES OF DERMATOLOGY, 1978, 114 (04) :531-535

[9] Effects of an interleukin-5 blocking monoclonal antibody on eosinophils, airway hyper-responsiveness, and the late asthmatic response [J].

Leckie, MJ ;

ten Brinke, A ;

Khan, J ;

Diamant, Z ;

O'Connor, BJ ;

Walls, CM ;

Mathur, AK ;

Cowley, HC ;

Chung, KF ;

Djukanovic, R ;

Hansel, TT ;

Holgate, T ;

Sterk, PJ ;

Barnes, PJ .

LANCET, 2000, 356 (9248) :2144-2148

[10] Intrinsic defect in T cell production of interleukin (IL)-13 in the absence of both IL-5 and eotaxin precludes the development of eosinophilia and airways hyperreactivity in experimental asthma [J].

Mattes, J ;

Yang, M ;

Mahalingam, S ;

Kuehr, J ;

Webb, DC ;

Simson, L ;

Hogan, SP ;

Koskinen, A ;

McKenzie, ANJ ;

Dent, LA ;

Rothenberg, ME ;

Matthaei, KI ;

Young, IG ;

Foster, PS .

JOURNAL OF EXPERIMENTAL MEDICINE, 2002, 195 (11) :1433-1444

← 1 2 →