Regulation and execution of programmed cell death in response to pathogens, stress and developmental cues

被引：152

作者：

Beers, EP ^{[1
]}

McDowell, JM

机构：

[1] Virginia Polytech Inst & State Univ, Dept Hort, Blacksburg, VA 24061 USA

[2] Virginia Polytech Inst & State Univ, Fralin Biotechnol Ctr, Blacksburg, VA 24061 USA

[3] Virginia Polytech Inst & State Univ, Dept Plant Pathol Physiol & Weed Sci, Blacksburg, VA 24061 USA

来源：

CURRENT OPINION IN PLANT BIOLOGY | 2001年 / 4卷 / 06期

基金：

美国国家卫生研究院;

关键词：

D O I：

10.1016/S1369-5266(00)00216-8

中图分类号：

Q94 [植物学];

学科分类号：

071001 ;

摘要：

Recent studies have expanded our view of the interactions between small molecule signals that regulate the hypersensitive response and other forms of cell suicide in plants. The mitochondrion has received increasing support as a mediator of at least some forms of programmed cell death in plants. In addition, new information provides a glimpse of how plant hormone signaling may be integrated with extensive autolysis, sensitivity to reactive oxygen intermediates and cell death.

引用

页码：561 / 567

页数：7

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