Production of doughnut-shaped, protease-defective particles from a human T cell clone carrying a provirus with specific mutations in the env, pol, vpr, and nef genes

被引:12
作者
Bahmani, MK
Kameoka, M
Nakaya, T
Fujinaga, K
Zhong, Q
Takahashi, H
Nakano, T
Nakai, M
Ueda, S
Jones, IM
Luftig, RB
Ikuta, K
机构
[1] HOKKAIDO UNIV,INST IMMUNOL SCI,SECT SEROL,KITA KU,SAPPORO,HOKKAIDO 060,JAPAN
[2] OSAKA MED COLL,TAKATSUKI,OSAKA 569,JAPAN
[3] OSAKA UNIV,MICROBIAL DIS RES INST,SUITA,OSAKA 565,JAPAN
[4] NERC,INST VIROL,OXFORD OX1 3SR,ENGLAND
[5] LOUISIANA STATE UNIV,MED CTR,DEPT MICROBIOL IMMUNOL & PARASITOL,NEW ORLEANS,LA 70112
关键词
D O I
10.1089/aid.1997.13.523
中图分类号
R392 [医学免疫学]; Q939.91 [免疫学];
学科分类号
100102 ;
摘要
[No abstract available]
引用
收藏
页码:523 / 526
页数:4
相关论文
共 16 条
[1]   PRODUCTION OF ACQUIRED IMMUNODEFICIENCY SYNDROME-ASSOCIATED RETROVIRUS IN HUMAN AND NONHUMAN CELLS TRANSFECTED WITH AN INFECTIOUS MOLECULAR CLONE [J].
ADACHI, A ;
GENDELMAN, HE ;
KOENIG, S ;
FOLKS, T ;
WILLEY, R ;
RABSON, A ;
MARTIN, MA .
JOURNAL OF VIROLOGY, 1986, 59 (02) :284-291
[2]  
AMEISEN JC, 1991, IMMUNOL TODAY, V12, P102
[3]   DEFECTIVE HUMAN IMMUNODEFICIENCY VIRUS (HIV) PARTICLES PRODUCED BY CLONED CELLS OF HTLV-I-CARRYING MT-4 CELLS PERSISTENTLY INFECTED WITH HIV [J].
IKUTA, K ;
MORITA, C ;
NAKAI, M ;
YAMAMOTO, N ;
KATO, S .
JAPANESE JOURNAL OF CANCER RESEARCH, 1988, 79 (04) :418-423
[4]   Protease-defective, gp120-containing human immunodeficiency virus type 1 particles induce apoptosis more efficiently than does wild-type virus or recombinant gp120 protein in healthy donor-derived peripheral blood T cells [J].
Kameoka, M ;
Kimura, T ;
Zheng, YH ;
Suzuki, S ;
Fujinaga, K ;
Luftig, RB ;
Ikuta, K .
JOURNAL OF CLINICAL MICROBIOLOGY, 1997, 35 (01) :41-47
[5]   High susceptibility of U937-derived subclones to human immunodeficiency virus type 1 infection correlates with accumulation of unintegrated circular viral DNA [J].
Kameoka, M ;
Kimura, T ;
Okada, Y ;
Fujinaga, K ;
Nakaya, T ;
Takahashi, H ;
Kishi, M ;
Ikuta, K .
VIRUS GENES, 1996, 12 (02) :117-129
[6]  
KAMEOKA M, 1996, INT IMMUNOL, V11, P1687
[7]   NATURALLY-OCCURRING ACCESSORY GENE-MUTATIONS LEAD TO PERSISTENT HUMAN-IMMUNODEFICIENCY-VIRUS TYPE-1 INFECTION OF CD4-POSITIVE T-CELLS [J].
KISHI, M ;
ZHENG, YH ;
BAHMANI, MK ;
TOKUNAGA, K ;
TAKAHASHI, H ;
KAKINUMA, M ;
LAI, PK ;
NONOYAMA, M ;
LUFTIG, RB ;
IKUTA, K .
JOURNAL OF VIROLOGY, 1995, 69 (12) :7507-7518
[8]  
LUFTIG RB, 1994, ASM NEWS, V60, P417
[9]   PERSISTENT INFECTION OF MT-4 CELLS BY HUMAN-IMMUNODEFICIENCY-VIRUS TYPE-1 BECOMES INCREASINGLY LIKELY WITH IN-VITRO SERIAL PASSAGE OF WILD-TYPE BUT NOT NEF MUTANT VIRUS [J].
NISHINO, Y ;
NAKAYA, T ;
FUJINAGA, K ;
KISHI, M ;
AZUMA, I ;
IKUTA, K .
JOURNAL OF GENERAL VIROLOGY, 1994, 75 :2241-2251
[10]   HUMAN-IMMUNODEFICIENCY-VIRUS TYPE-1 VIF, VPR, AND VPU MUTANTS CAN PRODUCE PERSISTENTLY INFECTED-CELLS [J].
NISHINO, Y ;
KISHI, M ;
SUMIYA, M ;
OGAWA, K ;
ADACHI, A ;
MAOTANIIMAI, K ;
KATO, S ;
HIRAI, K ;
IKUTA, K .
ARCHIVES OF VIROLOGY, 1991, 120 (3-4) :181-192