High glucose diets shorten lifespan of Caenorhabditis elegans via ectopic apoptosis induction

被引:39
作者
Choi, Shin Sik [1 ]
机构
[1] Myongji Univ, Dept Food & Nutr, Coll Nat Sci, Yongin 449728, Gyeonggi, South Korea
关键词
Glucose; lifespan; apoptosis; locomotion; aging; PROGRAMMED CELL-DEATH; C-ELEGANS; SILKWORM HEMOLYMPH; METABOLIC SYNDROME; GENETIC-CONTROL; GLYCEMIC INDEX; INHIBITION; DISEASE; CED-9;
D O I
10.4162/nrp.2011.5.3.214
中图分类号
R15 [营养卫生、食品卫生]; TS201 [基础科学];
学科分类号
100403 [营养与食品卫生学];
摘要
Diets based on carbohydrates increase rapidly the blood glucose level due to the fast conversion of carbohydrates to glucose. High glucose diets have been known to induce many lifestyle diseases. Here, we demonstrated that high glucose diet shortened the lifespan of Caenorhabditis elegans through apoptosis induction. Control adult groups without glucose diet lived for 30 days, whereas animals fed 10 mg/L of D-glucose lived only for 20 days. The reduction of lifespan by glucose diet showed a dose-dependent profile in the concentration range of glucose from 1 to 20 mg/L. Aging effect of high glucose diet was examined by measurement of response time for locomotion after stimulating movement of the animals by touching. Glucose diet decreased the locomotion capacity of the animals during mid-adulthood. High glucose diets also induced ectopic apoptosis in the body of C. elegans, which is a potent mechanism that can explain the shortened lifespan and aging. Apoptotic cell corpses stained with SYTO 12 were found in the worms fed 10 mg/L of glucose. Mutation of core apoptotic regulatory genes, CED-3 and CED-4, inhibited the reduction of viability induced by high glucose diet, which indicates that these regulators were required for glucose-induced apoptosis or lifespan shortening. Thus, we conclude that high glucose diets have potential for inducing ectopic apoptosis in the body, resulting in a shortened lifespan accompanied with loss of locomotion capacity.
引用
收藏
页码:214 / 218
页数:5
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