Chronic myeloid leukemia

[1] Regulation of the oncogenic activity of BCR-ABL by a tightly bound substrate protein RIN1 [J]. IMMUNITY, 1997, 6 (06) : 773 - 782

[2] DIFFERENTIAL COMPLEMENTATION OF BCR-ABL POINT MUTANTS WITH C-MYC [J]. SCIENCE, 1994, 264 (5157) : 424 - 426

[3] THE SPECTRUM OF MOLECULAR ALTERATIONS IN THE EVOLUTION OF CHRONIC MYELOCYTIC-LEUKEMIA [J]. JOURNAL OF CLINICAL INVESTIGATION, 1991, 87 (06) : 2042 - 2047

[4] Primary proliferating immature myeloid cells from CML patients are not resistant to induction of apoptosis by DNA damage and growth factor withdrawal [J]. BRITISH JOURNAL OF HAEMATOLOGY, 1996, 95 (03) : 501 - 507

[5] UK MEDICAL-RESEARCH-COUNCIL RANDOMIZED, MULTICENTER TRIAL OF INTERFERON-ALPHA-N1 FOR CHRONIC MYELOID-LEUKEMIA - IMPROVED SURVIVAL IRRESPECTIVE OF CYTOGENETIC RESPONSE [J]. LANCET, 1995, 345 (8962) : 1392 - 1397

[6] APOPTOSIS IN CHRONIC MYELOID-LEUKEMIA - NORMAL RESPONSES BY PROGENITOR CELLS TO GROWTH-FACTOR DEPRIVATION, X-IRRADIATION AND GLUCOCORTICOIDS [J]. BRITISH JOURNAL OF HAEMATOLOGY, 1995, 91 (02) : 387 - 393

[7] ARCESE W, 1993, BLOOD, V82, P3211

[8] BARNETT MJ, 1994, BLOOD, V84, P724

[9] Bcr/Abl expression stimulates integrin function in hematopoietic cell lines [J]. JOURNAL OF CLINICAL INVESTIGATION, 1996, 98 (02) : 521 - 528

[10] PROLONGED ADMINISTRATION OF INTERFERON-ALPHA IN PATIENTS WITH CHRONIC-PHASE PHILADELPHIA-CHROMOSOME-POSITIVE CHRONIC MYELOGENOUS LEUKEMIA BEFORE ALLOGENEIC BONE-MARROW TRANSPLANTATION MAY ADVERSELY AFFECT TRANSPLANT OUTCOME [J]. BLOOD, 1995, 85 (10) : 2981 - 2990

[1] Regulation of the oncogenic activity of BCR-ABL by a tightly bound substrate protein RIN1 [J]. IMMUNITY, 1997, 6 (06) : 773 - 782

[2] DIFFERENTIAL COMPLEMENTATION OF BCR-ABL POINT MUTANTS WITH C-MYC [J]. SCIENCE, 1994, 264 (5157) : 424 - 426

[3] THE SPECTRUM OF MOLECULAR ALTERATIONS IN THE EVOLUTION OF CHRONIC MYELOCYTIC-LEUKEMIA [J]. JOURNAL OF CLINICAL INVESTIGATION, 1991, 87 (06) : 2042 - 2047

[4] Primary proliferating immature myeloid cells from CML patients are not resistant to induction of apoptosis by DNA damage and growth factor withdrawal [J]. BRITISH JOURNAL OF HAEMATOLOGY, 1996, 95 (03) : 501 - 507

[5] UK MEDICAL-RESEARCH-COUNCIL RANDOMIZED, MULTICENTER TRIAL OF INTERFERON-ALPHA-N1 FOR CHRONIC MYELOID-LEUKEMIA - IMPROVED SURVIVAL IRRESPECTIVE OF CYTOGENETIC RESPONSE [J]. LANCET, 1995, 345 (8962) : 1392 - 1397

[6] APOPTOSIS IN CHRONIC MYELOID-LEUKEMIA - NORMAL RESPONSES BY PROGENITOR CELLS TO GROWTH-FACTOR DEPRIVATION, X-IRRADIATION AND GLUCOCORTICOIDS [J]. BRITISH JOURNAL OF HAEMATOLOGY, 1995, 91 (02) : 387 - 393

[7] ARCESE W, 1993, BLOOD, V82, P3211

[8] BARNETT MJ, 1994, BLOOD, V84, P724

[9] Bcr/Abl expression stimulates integrin function in hematopoietic cell lines [J]. JOURNAL OF CLINICAL INVESTIGATION, 1996, 98 (02) : 521 - 528

[10] PROLONGED ADMINISTRATION OF INTERFERON-ALPHA IN PATIENTS WITH CHRONIC-PHASE PHILADELPHIA-CHROMOSOME-POSITIVE CHRONIC MYELOGENOUS LEUKEMIA BEFORE ALLOGENEIC BONE-MARROW TRANSPLANTATION MAY ADVERSELY AFFECT TRANSPLANT OUTCOME [J]. BLOOD, 1995, 85 (10) : 2981 - 2990