Yin and yang of MCP-1

被引：21

作者：

Becker, LC ^{[1
]}

机构：

[1] Johns Hopkins Univ, Sch Med, Dept Med, Div Cardiol, Baltimore, MD 21205 USA

来源：

CIRCULATION RESEARCH | 2005年 / 96卷 / 08期

关键词：

monocyte chemoattractant protein-1; ischemia; myocardial infarction; inflammation; left ventricular remodeling; matrix metalloproteinases;

D O I：

10.1161/01.RES.0000165652.82726.d9

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

引用

页码：812 / 814

页数：3

共 20 条

[1] MINIMALLY MODIFIED LOW-DENSITY-LIPOPROTEIN INDUCES MONOCYTE CHEMOTACTIC PROTEIN-1 IN HUMAN ENDOTHELIAL-CELLS AND SMOOTH-MUSCLE CELLS [J].

CUSHING, SD ;

BERLINER, JA ;

VALENTE, AJ ;

TERRITO, MC ;

NAVAB, M ;

PARHAMI, F ;

GERRITY, R ;

SCHWARTZ, CJ ;

FOGELMAN, AM .

PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1990, 87 (13) :5134-5138

[2]

Dawson TC, 1999, ATHEROSCLEROSIS, V143, P205

[3] CCL2/monocyte chemoattractant protein-1 regulates inflammatory responses critical to healing myocardial infarcts [J].

Dewald, O ;

Zymek, P ;

Winkelmann, K ;

Koerting, A ;

Ren, GF ;

Abou-Khamis, T ;

Michael, LH ;

Rollins, BJ ;

Entman, ML ;

Frangogiannis, NG .

CIRCULATION RESEARCH, 2005, 96 (08) :881-889

[4] Costimulation of fibroblast collagen and transforming growth factor beta(1) gene expression by monocyte chemoattractant protein-1 via specific receptors [J].

GharaeeKermani, M ;

Denholm, EM ;

Phan, SH .

JOURNAL OF BIOLOGICAL CHEMISTRY, 1996, 271 (30) :17779-17784

[5] Absence of monocyte chemoattractant protein-1 reduces atherosclerosis in low density lipoprotein receptor-deficient mice [J].

Gu, L ;

Okada, Y ;

Clinton, SK ;

Gerard, C ;

Sukhova, GK ;

Libby, P ;

Rollins, BJ .

MOLECULAR CELL, 1998, 2 (02) :275-281

[6] Chemokine receptor CCR2 expression and monocyte chemoattractant protein-1-mediated chemotaxis in human monocytes - A regulatory role for plasma LDL [J].

Han, KH ;

Tangirala, RK ;

Green, SR ;

Quehenberger, O .

ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY, 1998, 18 (12) :1983-1991

[7] G-CSF prevents cardiac remodeling after myocardial infarction by activating the Jak-Stat pathway in cardiomyocytes [J].

Harada, M ;

Qin, YJ ;

Takano, H ;

Minamino, T ;

Zou, YZ ;

Toko, H ;

Ohtsuka, M ;

Matsuura, K ;

Sano, M ;

Nishi, J ;

Iwanaga, K ;

Akazawa, H ;

Kunieda, T ;

Zhu, WD ;

Hasegawa, H ;

Kunisada, K ;

Nagai, T ;

Nakaya, H ;

Yamauchi-Takihara, K ;

Komuro, I .

NATURE MEDICINE, 2005, 11 (03) :305-311

[8] Anti-monocyte chemoattractant protein-1 gene therapy attenuates left ventricular remodeling and failure after experimental myocardial infarction [J].

Hayashidani, S ;

Tsutsui, H ;

Shiomi, T ;

Ikeuchi, M ;

Matsusaka, H ;

Suematsu, N ;

Wen, J ;

Egashira, K ;

Takeshita, A .

CIRCULATION, 2003, 108 (17) :2134-2140

[9] Accelerated LV remodeling after myocardial infarction in TIMP-1-deficient mice: effects of exogenous MMP inhibition [J].

Ikonomidis, JS ;

Hendrick, JW ;

Parkhurst, AM ;

Herron, AR ;

Escobar, PG ;

Dowdy, KB ;

Stroud, RE ;

Hapke, E ;

Zile, MR ;

Spinale, FG .

AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY, 2005, 288 (01) :H149-H158

[10]

JIANG YL, 1992, J IMMUNOL, V148, P2423

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