Nitric oxide synthesis in atherosclerosis

[1] NITRIC-OXIDE AND PROSTACYCLIN - DIVERGENCE OF INHIBITORY EFFECTS ON MONOCYTE CHEMOTAXIS AND ADHESION TO ENDOTHELIUM INVITRO [J]. ARTERIOSCLEROSIS AND THROMBOSIS, 1991, 11 (02): : 254 - 260

[2] CHRONIC BLOCKADE OF NITRIC-OXIDE SYNTHESIS IN THE RAT PRODUCES SYSTEMIC HYPERTENSION AND GLOMERULAR DAMAGE [J]. JOURNAL OF CLINICAL INVESTIGATION, 1992, 90 (01) : 278 - 281

[3] COMPARISON OF RESPONSES TO ACETYLCHOLINE AND SEROTONIN ON ISOLATED CANINE AND HUMAN CORONARY-ARTERIES [J]. CARDIOVASCULAR RESEARCH, 1989, 23 (09) : 780 - 787

[4] BIRNBAUMER L, 1990, ANNU REV PHARMACOL, V30, P675

[5] CHRONIC EXPOSURE OF CULTURED ENDOTHELIAL-CELLS TO EICOSAPENTAENOIC ACID POTENTIATES THE RELEASE OF ENDOTHELIUM-DERIVED RELAXING FACTOR(S) [J]. BRITISH JOURNAL OF PHARMACOLOGY, 1990, 99 (01) : 176 - 180

[6] BUSSE R, 1993, CIRCULATION, V87, P18

[7] Buttery LDK, 1996, LAB INVEST, V75, P77

[8] INVESTIGATION OF DECREASED AVAILABILITY OF NITRIC-OXIDE PRECURSOR AS THE MECHANISM RESPONSIBLE FOR IMPAIRED ENDOTHELIUM-DEPENDENT VASODILATION IN HYPERCHOLESTEROLEMIC PATIENTS [J]. JOURNAL OF THE AMERICAN COLLEGE OF CARDIOLOGY, 1994, 23 (04) : 844 - 850

[9] NONINVASIVE DETECTION OF ENDOTHELIAL DYSFUNCTION IN CHILDREN AND ADULTS AT RISK OF ATHEROSCLEROSIS [J]. LANCET, 1992, 340 (8828) : 1111 - 1115

[10] L-ARGININE ABROGATES SALT-SENSITIVE HYPERTENSION IN DAHL RAPP RATS [J]. JOURNAL OF CLINICAL INVESTIGATION, 1991, 88 (05) : 1559 - 1567

[1] NITRIC-OXIDE AND PROSTACYCLIN - DIVERGENCE OF INHIBITORY EFFECTS ON MONOCYTE CHEMOTAXIS AND ADHESION TO ENDOTHELIUM INVITRO [J]. ARTERIOSCLEROSIS AND THROMBOSIS, 1991, 11 (02): : 254 - 260

[2] CHRONIC BLOCKADE OF NITRIC-OXIDE SYNTHESIS IN THE RAT PRODUCES SYSTEMIC HYPERTENSION AND GLOMERULAR DAMAGE [J]. JOURNAL OF CLINICAL INVESTIGATION, 1992, 90 (01) : 278 - 281

[3] COMPARISON OF RESPONSES TO ACETYLCHOLINE AND SEROTONIN ON ISOLATED CANINE AND HUMAN CORONARY-ARTERIES [J]. CARDIOVASCULAR RESEARCH, 1989, 23 (09) : 780 - 787

[4] BIRNBAUMER L, 1990, ANNU REV PHARMACOL, V30, P675

[5] CHRONIC EXPOSURE OF CULTURED ENDOTHELIAL-CELLS TO EICOSAPENTAENOIC ACID POTENTIATES THE RELEASE OF ENDOTHELIUM-DERIVED RELAXING FACTOR(S) [J]. BRITISH JOURNAL OF PHARMACOLOGY, 1990, 99 (01) : 176 - 180

[6] BUSSE R, 1993, CIRCULATION, V87, P18

[7] Buttery LDK, 1996, LAB INVEST, V75, P77

[8] INVESTIGATION OF DECREASED AVAILABILITY OF NITRIC-OXIDE PRECURSOR AS THE MECHANISM RESPONSIBLE FOR IMPAIRED ENDOTHELIUM-DEPENDENT VASODILATION IN HYPERCHOLESTEROLEMIC PATIENTS [J]. JOURNAL OF THE AMERICAN COLLEGE OF CARDIOLOGY, 1994, 23 (04) : 844 - 850

[9] NONINVASIVE DETECTION OF ENDOTHELIAL DYSFUNCTION IN CHILDREN AND ADULTS AT RISK OF ATHEROSCLEROSIS [J]. LANCET, 1992, 340 (8828) : 1111 - 1115

[10] L-ARGININE ABROGATES SALT-SENSITIVE HYPERTENSION IN DAHL RAPP RATS [J]. JOURNAL OF CLINICAL INVESTIGATION, 1991, 88 (05) : 1559 - 1567