Alveolar proteinosis: a disease of mice and men

被引:3
作者
Carraway, MS [1 ]
Piantadosi, CA
Wright, JR
机构
[1] Duke Univ, Med Ctr, Dept Med, Durham, NC 27710 USA
[2] Duke Univ, Med Ctr, Dept Cell Biol, Durham, NC 27710 USA
关键词
D O I
10.1152/ajplung.2001.280.3.L377
中图分类号
Q4 [生理学];
学科分类号
071003 ;
摘要
引用
收藏
页码:L377 / L378
页数:2
相关论文
共 14 条
[1]   GM-CSF and GM-CSF βc receptor in adult patients with pulmonary alveolar proteinosis [J].
Bewig, B ;
Wang, XD ;
Kirsten, D ;
Dalhoff, K ;
Schäfer, H .
EUROPEAN RESPIRATORY JOURNAL, 2000, 15 (02) :350-357
[2]   Detection of granulocyte-macrophage colony-stimulating factor in patients with pulmonary alveolar proteinosis [J].
Carraway, MS ;
Ghio, AJ ;
Carter, JD ;
Piantadosi, CA .
AMERICAN JOURNAL OF RESPIRATORY AND CRITICAL CARE MEDICINE, 2000, 161 (04) :1294-1299
[3]   INVOLVEMENT OF GRANULOCYTE-MACROPHAGE COLONY-STIMULATING FACTOR IN PULMONARY HOMEOSTASIS [J].
DRANOFF, G ;
CRAWFORD, AD ;
SADELAIN, M ;
REAM, B ;
RASHID, A ;
BRONSON, RT ;
DICKERSIN, GR ;
BACHURSKI, CJ ;
MARK, EL ;
WHITSETT, JA ;
MULLIGAN, RC .
SCIENCE, 1994, 264 (5159) :713-716
[4]   Surfactant metabolism in transgenic mice after granulocyte macrophage-colony stimulating factor ablation [J].
Ikegami, M ;
Ueda, T ;
Hull, W ;
Whitsett, JA ;
Mulligan, RC ;
Dranoff, G ;
Jobe, AH .
AMERICAN JOURNAL OF PHYSIOLOGY-LUNG CELLULAR AND MOLECULAR PHYSIOLOGY, 1996, 270 (04) :L650-L658
[5]   PULMONARY ALVEOLAR PROTEINOSIS - PROSPECTIVE CLINICAL-EXPERIENCE IN 23 PATIENTS FOR 15 YEARS [J].
KARIMAN, K ;
KYLSTRA, JA ;
SPOCK, A .
LUNG, 1984, 162 (04) :223-231
[6]   Exogenous granulocyte-macrophage colony-stimulating factor administration for pulmonary alveolar proteinosis [J].
Kavuru, MS ;
Sullivan, EJ ;
Piccin, R ;
Thomassen, MJ ;
Stoller, JK .
AMERICAN JOURNAL OF RESPIRATORY AND CRITICAL CARE MEDICINE, 2000, 161 (04) :1143-1148
[7]   Idiopathic pulmonary alveolar proteinosis as an autoimmune disease with neutralizing antibody against granulocyte/macrophage colony-stimulating factor [J].
Kitamura, T ;
Tanaka, N ;
Watanabe, J ;
Uchida, K ;
Kanegasaki, S ;
Yamada, Y ;
Nakata, K .
JOURNAL OF EXPERIMENTAL MEDICINE, 1999, 190 (06) :875-880
[8]   The biological consequences of excess GM-CSF levels in transgenic mice also lacking high-affinity receptors for GM-CSF [J].
Metcalf, D ;
Mifsud, S ;
Di Rago, L ;
Robb, L ;
Nicola, NA ;
Alexander, W .
LEUKEMIA, 1998, 12 (03) :353-362
[9]   MICE DEFICIENT FOR THE IL-3/GM-CSF/IL-5 BETA-C RECEPTOR EXHIBIT LUNG PATHOLOGY AND IMPAIRED IMMUNE-RESPONSE, WHILE BETA-IL3 RECEPTOR-DEFICIENT MICE ARE NORMAL [J].
NISHINAKAMURA, R ;
NAKAYAMA, N ;
HIRABAYASHI, Y ;
INOUE, T ;
AUD, D ;
MCNEIL, T ;
AZUMA, S ;
YOSHIDA, S ;
TOYODA, Y ;
ARAI, K ;
MIYAJIMA, A ;
MURRAY, R .
IMMUNITY, 1995, 2 (03) :211-222
[10]  
Seymour JF, 1998, BLOOD, V92, P2657