Role of apoptosis in alcoholic liver injury

被引:36
作者
Ishii, H
Adachi, M
Fernández-Checa, JC
Cederbaum, AI
Deaciuc, IV
Nanji, AA
机构
[1] Keio Univ, Sch Med, Dept Internal Med, Shinjuku Ku, Tokyo 1608582, Japan
[2] Univ Penn, Med Ctr, Dept Pathol & Lab Med, Philadelphia, PA 19104 USA
[3] CSIC, Hosp Clin & Prov, IDIBAPS, Barcelona, Spain
[4] CUNY Mt Sinai Sch Med, Dept Biochem, New York, NY 10029 USA
[5] Univ Kentucky, GI Div, Lexington, KY USA
关键词
apoptosis; alcoholic liver disease; oxidative stress; glutathione; CYP2E1;
D O I
10.1097/01.alc.0000086766.46408.bf
中图分类号
R194 [卫生标准、卫生检查、医药管理];
学科分类号
摘要
This article represents the proceedings of a symposium at the 2002 ISBRA/RSA meeting in San Francisco, USA. The organizer and chair was H. Ishii and co-chair was A.A Nanji. The presentations were (1) Introduction: Apoptosis in alcoholic liver disease, by A.A. Nanji; (2) Mitochondria, oxidative stress and apoptosis in alcoholic liver disease, by M. Adachi; (3) Regulation of cell death by mitochondrial glutathione, by J.C. Ferndndez-Checa; (4) Toxicity of ethanol in HepG2 cells that express CYP2E1, by A.I.. Cederbaum; (5) Is alcohol-enhanced liver apoptosis a pathogenic factor in alcoholic liver disease? by I.V. Deaciuc.
引用
收藏
页码:1207 / 1212
页数:6
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