Structural basis for the recruitment of profilin-actin complexes during filament elongation by Ena/VASP

被引:199
作者
Ferron, Francois [1 ]
Rebowski, Grzegorz [1 ]
Lee, Sung Haeng [1 ]
Dominguez, Roberto [1 ]
机构
[1] Univ Penn, Sch Med, Dept Physiol, Philadelphia, PA 19104 USA
关键词
actin; cytoskeleton dynamics; filament elongation; profilin;
D O I
10.1038/sj.emboj.7601874
中图分类号
Q5 [生物化学]; Q7 [分子生物学];
学科分类号
071010 ; 081704 ;
摘要
Cells sustain high rates of actin filament elongation by maintaining a large pool of actin monomers above the critical concentration for polymerization. Profilin-actin complexes constitute the largest fraction of polymerization-competent actin monomers. Filament elongation factors such as Ena/VASP and formin catalyze the transition of profilin-actin from the cellular pool onto the barbed end of growing filaments. The molecular bases of this process are poorly understood. Here we present structural and energetic evidence for two consecutive steps of the elongation mechanism: the recruitment of profilin-actin by the last poly-Pro segment of vasodilator-stimulated phosphoprotein (VASP) and the binding of profilin-actin simultaneously to this poly-Pro and to the G-actin-binding (GAB) domain of VASP. The actin monomer bound at the GAB domain is proposed to be in position to join the barbed end of the growing filament concurrently with the release of profilin.
引用
收藏
页码:4597 / 4606
页数:10
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