Science, medicine, and the future - Parkinson's disease

被引：142

作者：

Schapira, AHV ^{[1
]}

机构：

[1] Royal Free & Univ Coll Med Sch, Univ Dept Clin Neurosci, London, England

[2] UCL, Neurol Inst, London, England

来源：

BRITISH MEDICAL JOURNAL | 1999年 / 318卷 / 7179期

关键词：

D O I：

10.1136/bmj.318.7179.311

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

引用

页码：311 / 314

页数：4

共 17 条

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[3] GOLBE LI, 1995, ETIOLOGY PARKINSONS, P115
[4] Mitochondrial DNA transmission of the mitochondrial defect in Parkinson's disease
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Schapira, AHV
[J]. ANNALS OF NEUROLOGY, 1998, 44 (02) : 177 - 186
[5] Glial cells and inflammation in Parkinson's disease: A role in neurodegeneration?
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[J]. ANNALS OF NEUROLOGY, 1998, 44 (03) : S115 - S120
[6] Mutations in the parkin gene cause autosomal recessive juvenile parkinsonism
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[J]. NATURE, 1998, 392 (6676) : 605 - 608
[7] Ala30Pro mutation in the gene encoding α-synuclein in Parkinson's disease
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[J]. NATURE GENETICS, 1998, 18 (02) : 106 - 108
[8] Electrical stimulation of the subthalamic nucleus in advanced Parkinson's disease
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[J]. NEW ENGLAND JOURNAL OF MEDICINE, 1998, 339 (16) : 1105 - 1111
[9] Chronic levodopa is not toxic for remaining dopamine neurons, but instead promotes their recovery, in rats with moderate nigrostriatal lesions
Murer, MG
Dziewczapolski, G
Menalled, LB
Garcia, MC
Agid, Y
Gershanik, O
Raisman-Vozari, R
[J]. ANNALS OF NEUROLOGY, 1998, 43 (05) : 561 - 575
[10] Mytilineou C, 1997, J NEUROCHEM, V68, P434

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