ST segment elevation induced by Class IC antiarrhythmic agents: Underlying electrophysiologic mechanisms and insights into drug-induced proarrhythmia

被引：56

作者：

Krishnan, S ^{[1
]}

Josephson, ME ^{[1
]}

机构：

[1] Beth Israel Deaconess Med Ctr, Harvard Thorndike Electrophysiol Inst, Boston, MA 02215 USA

来源：

JOURNAL OF CARDIOVASCULAR ELECTROPHYSIOLOGY | 1998年 / 9卷 / 11期

关键词：

encainide; flecainide; ST segment elevation; proarrhythmia;

D O I：

10.1111/j.1540-8167.1998.tb00088.x

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

Three patients in whom Class IC sodium channel blockers induced ST segment elevation in leads V-1 through V-3 are described. The underlying electrophysiologic mechanism, implications for drug-induced proarrhythmia, and the relationship of the finding to the Brugada syndrome type of idiopathic ventricular fibrillation are discussed.

引用

页码：1167 / 1172

页数：6

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