Role of PKCs and NF-κB activation in myocardial inflammation:: Enemy or ally?

被引：19

作者：

Abe, Jun-ichi ^{[1
]}

机构：

[1] Univ Rochester, Sch Med & Dent, Cardiovasc Res Inst, Rochester, NY 14642 USA

来源：

JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY | 2007年 / 43卷 / 04期

关键词：

PROTEIN-KINASE-C; REPERFUSION INJURY; CARDIAC MYOCYTE; ANGIOTENSIN-II; DELTA-PKC; CYCLIN D1; APOPTOSIS; EPSILON; BETA; ISCHEMIA;

D O I：

10.1016/j.yjmcc.2007.07.002

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

引用

页码：404 / 408

页数：5

共 44 条

[1] Protein kinase activation and myocardial ischemia/reperfusion injury [J].

Armstrong, SC .

CARDIOVASCULAR RESEARCH, 2004, 61 (03) :427-436

[2] Protein kinase Cε interacts with and inhibits the permeability transition pore in cardiac mitochondria [J].

Baines, CP ;

Song, CX ;

Zheng, YT ;

Wang, GW ;

Zhang, J ;

Wang, OL ;

Guo, Y ;

Bolli, R ;

Cardwell, EM ;

Ping, PP .

CIRCULATION RESEARCH, 2003, 92 (08) :873-880

[3] Control of apoptosis by Rel/NF-κB transcription factors [J].

Barkett, M ;

Gilmore, TD .

ONCOGENE, 1999, 18 (49) :6910-6924

[4]

Braz J. C., 2004, NAT MED, V15, P15

[5] Cardioprotective mechanisms of PKC isozyme-selective activators and inhibitors in the treatment of ischemia-reperfusion injury [J].

Budas, Grant R. ;

Churchill, Eric N. ;

Mochly-Rosen, Daria .

PHARMACOLOGICAL RESEARCH, 2007, 55 (06) :523-536

[6] The Rel/NF-κB family directly activates expression of the apoptosis inhibitor Bcl-xL [J].

Chen, CL ;

Edelstein, LC ;

Gélinas, C .

MOLECULAR AND CELLULAR BIOLOGY, 2000, 20 (08) :2687-2695

[7] Translocation of δPKC to mitochondria during cardiac reperfusion enhances superoxide anion production and induces loss in mitochondrial function [J].

Churchill, EN ;

Szweda, LI .

ARCHIVES OF BIOCHEMISTRY AND BIOPHYSICS, 2005, 439 (02) :194-199

[8] Reperfusion-induced translocation of δPKC to cardiac mitochondria prevents pyruvate dehydrogenase reactivation [J].

Churchill, EN ;

Murriel, CL ;

Chen, CH ;

Mochly-Rosen, D ;

Szweda, LI .

CIRCULATION RESEARCH, 2005, 97 (01) :78-85

[9] The ε subtype of protein kinase C is required for cardiomyocyte connexin-43 phosphorylation [J].

Doble, BW ;

Ping, PP ;

Kardami, E .

CIRCULATION RESEARCH, 2000, 86 (03) :293-301

[10] INTERLEUKIN-1 AND TUMOR NECROSIS FACTOR INHIBIT CARDIAC MYOCYTE BETA-ADRENERGIC RESPONSIVENESS [J].

GULICK, T ;

CHUNG, MK ;

PIEPER, SJ ;

LANGE, LG ;

SCHREINER, GF .

PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1989, 86 (17) :6753-6757

← 1 2 3 4 5 →