Tolcapone and neurotoxicity in Parkinson's disease

被引:10
作者
Kuhn, W [1 ]
Woitalla, D
Gerlach, M
Russ, H
Müller, T
机构
[1] Ruhr Univ Bochum, St Josef Hosp, Dept Neurol, D-44791 Bochum, Germany
[2] Univ Regensburg, Dept Neurol, D-8400 Regensburg, Germany
[3] Univ Wurzburg, Dept Neurochem, Wurzburg, Germany
关键词
D O I
10.1016/S0140-6736(05)70527-4
中图分类号
R5 [内科学];
学科分类号
1002 ; 100201 ;
摘要
引用
收藏
页码:1313 / 1314
页数:2
相关论文
共 5 条
[1]   SUBSTANTIA-NIGRA DEGENERATION AND TYROSINE-HYDROXYLASE DEPLETION CAUSED BY EXCESS S-ADENOSYLMETHIONINE IN THE RAT-BRAIN - SUPPORT FOR AN EXCESS METHYLATION HYPOTHESIS FOR PARKINSONISM [J].
CHARLTON, CG ;
MACK, J .
MOLECULAR NEUROBIOLOGY, 1994, 9 (1-3) :149-161
[2]  
DAPRADA M, 1994, CLIN NEUROPHARMACOL, V17, pS26
[3]   Oxidative damage caused by free radicals produced during catecholamine autoxidation: Protective effects of O-methylation and melatonin [J].
Miller, JW ;
Selhub, J ;
Joseph, JA .
FREE RADICAL BIOLOGY AND MEDICINE, 1996, 21 (02) :241-249
[4]   Catechol-O-methyltransferase inhibitors in Parkinson's disease [J].
Xie, T ;
Ho, SL ;
Ramsden, D .
LANCET, 1998, 351 (9120) :1966-1966
[5]  
ZURCHER G, 1991, NEW DEV THERAPY PARK, P37