Roles of complex gangliosides at the neuromuscular junction

被引：9

作者：

Bullen, RWM

O'Hanlon, GM

Wagner, E

Molenaar, PC

Furukawa, K

Plomp, JJ

Willison, HJ

机构：

[1] Leiden Univ, Ctr Med, Dept Neurophysiol, NL-2300 RC Leiden, Netherlands

[2] Leiden Univ, Ctr Med, Dept Neurol, NL-2300 RC Leiden, Netherlands

[3] Univ Glasgow, So Gen Hosp, Dept Neurol, Glasgow, Lanark, Scotland

[4] Nagoya Univ, Sch Med, Dept Biochem, Nagoya, Aichi, Japan

来源：

MYASTHENIA GRAVIS AND RELATED DISORDERS: BIOMCHEMICAL BASIS FOR DISEASE OF THE NEUROMUSCULAR JUNCTION | 2003年 / 998卷

关键词：

complex gangliosides; neuromuscular junction (NMJ); Miller Fisher syndrome (MFS);

D O I：

10.1196/annals.1254.051

中图分类号：

Q5 [生物化学]; Q7 [分子生物学];

学科分类号：

071010 ; 081704 ;

摘要：

引用

页码：401 / 403

页数：3

共 4 条

[1]

Bullens RWM, 2002, J NEUROSCI, V22, P6876

[2]

Plomp JJ, 1999, ANN NEUROL, V45, P189, DOI 10.1002/1531-8249(199902)45:2<189::AID-ANA9>3.0.CO

[3]

2-T

[4] Mice with disrupted GM2/GD2 synthase gene lack complex gangliosides but exhibit only subtle defects in their nervous system [J].

Takamiya, K ;

Yamamoto, A ;

Furukawa, K ;

Yamashiro, S ;

Shin, M ;

Okada, M ;

Fukumoto, S ;

Haraguchi, M ;

Takeda, N ;

Fujimura, K ;

Sakae, M ;

Kishikawa, M ;

Shiku, H ;

Furukawa, K ;

Aizawa, S .

PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1996, 93 (20) :10662-10667

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