The continuing saga of complement receptor type 3 (CR3)

被引：38

作者：

Todd, RF

机构：

来源：

JOURNAL OF CLINICAL INVESTIGATION | 1996年 / 98卷 / 01期

关键词：

D O I：

10.1172/JCI118752

中图分类号：

R-3 [医学研究方法]; R3 [基础医学];

学科分类号：

1001 ;

摘要：

引用

收藏

页码：1 / 2

页数：2

相关论文

共 6 条

[1] ARNAOUT MA, 1990, BLOOD, V75, P1037
[2] THE I-DOMAIN IS A MAJOR RECOGNITION SITE ON THE LEUKOCYTE INTEGRIN MAC-1 (CD11B/CD18) FOR 4 DISTINCT ADHESION LIGANDS
DIAMOND, MS
GARCIAAGUILAR, J
BICKFORD, JK
CORBI, AL
SPRINGER, TA
[J]. JOURNAL OF CELL BIOLOGY, 1993, 120 (04) : 1031 - 1043
[3] Integrins as promiscuous signal transduction devices
Petty, HR
Todd, RF
[J]. IMMUNOLOGY TODAY, 1996, 17 (05): : 209 - 212
[4] SPECIFICITY OF MEMBRANE COMPLEMENT RECEPTOR TYPE 3 (CR3) FOR BETA-GLUCANS
ROSS, GD
CAIN, JA
MYONES, BL
NEWMAN, SL
LACHMANN, PJ
[J]. COMPLEMENT, 1987, 4 (02): : 61 - 74
[5] Thornton BP, 1996, J IMMUNOL, V156, P1235
[6] Soluble beta-glucan polysaccharide binding to the lectin site of neutrophil or natural killer cell complement receptor type 3 (CD11b/CD18) generates a primed state of the receptor capable of mediating cytotoxicity of iC3b-opsonized target cells
Vetvicka, V
Thornton, BP
Ross, GD
[J]. JOURNAL OF CLINICAL INVESTIGATION, 1996, 98 (01) : 50 - 61