Recipient T cells mediate reperfusion injury after lung transplantation in the rat

被引:76
作者
de Perrot, M
Young, K
Imai, Y
Liu, MY
Waddell, TK
Fischer, S
Zhang, L
Keshavjee, S
机构
[1] Univ Toronto, Toronto Gen Hosp, Thorac Surg Res Lab, Toronto, ON M5G 2C4, Canada
[2] Univ Toronto, Toronto Gen Hosp, Dept Lab Med, Toronto, ON M5G 2C4, Canada
[3] Univ Toronto, Toronto Gen Hosp, Dept Pathobiol, Toronto, ON M5G 2C4, Canada
[4] Univ Toronto, Toronto Gen Hosp, Dept Immunol, Toronto, ON M5G 2C4, Canada
关键词
D O I
10.4049/jimmunol.171.10.4995
中图分类号
R392 [医学免疫学]; Q939.91 [免疫学];
学科分类号
100102 ;
摘要
Leukocytes have been implicated in ischemia-reperfusion (IR) injury of the lung, but the individual role of T cells has not been explored. Recent evidence in mice suggests that T cells may play a role in IR injury. Using a syngeneic (Lewis to Lewis) rat lung transplant model, we observed that recipient CD4(+) T cells infiltrated lung grafts within 1 h of reperfusion and up-regulated the expression of CD25 over the ensuing 12 h. Nude rats (rnu/rnu) and heterozygous rats (rnu/+) were used to determine the role of T cells in IR injury. No significant difference in lung function was observed between nude and heterozygous recipient rats after 2 h of reperfusion. However, after 12 h of reperfusion, recipient nude rats had significantly higher oxygenation and lower peak airway pressure than recipient heterozygous rats. This was associated with significantly lower levels of IFN-gamma in transplanted lung tissue of recipient nude rats. Reconstitution of recipient nude rats with T cells from heterozygous rats restored IR injury after 12 h of reperfusion. The effect of T cells was independent of neutrophil recruitment and activation in the transplanted lung. These results demonstrate that recipient T cells are activated and mediate IR injury during lung transplantation in rats.
引用
收藏
页码:4995 / 5002
页数:8
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