Disorders of lung matrix remodeling

被引：177

作者：

Chapman, HA

机构：

[1] Univ Calif San Francisco, Div Pulm & Crit Care, Dept Med, San Francisco, CA 94143 USA

[2] Univ Calif San Francisco, Cardiovasc Res Inst, San Francisco, CA 94143 USA

来源：

JOURNAL OF CLINICAL INVESTIGATION | 2004年 / 113卷 / 02期

关键词：

D O I：

10.1172/JCI200420729

中图分类号：

R-3 [医学研究方法]; R3 [基础医学];

学科分类号：

1001 [基础医学];

摘要：

A set of lung diseases share the tendency for the development of progressive fibrosis ultimately leading to respiratory failure. This review examines the common pathogenetic features of these disorders in light of recent observations in both humans and animal models of disease, which reveal important pathways of lung matrix remodeling.

引用

收藏

页码：148 / 157

页数：10

相关论文

共 103 条

[1]

DRUG-INDUCED PULMONARY FIBROSIS [J].

ADAMSON, IYR .

ENVIRONMENTAL HEALTH PERSPECTIVES, 1984, 55 (APR) :25-36

[2]

Fas and fas ligand are up-regulated in pulmonary edema fluid and lung tissue of patients with acute lung injury and the acute respiratory distress syndrome [J].

Albertine, KH ;

Soulier, MF ;

Wang, ZM ;

Ishizaka, A ;

Hashimoto, S ;

Zimmerman, GA ;

Matthay, MA ;

Ware, LB .

AMERICAN JOURNAL OF PATHOLOGY, 2002, 161 (05) :1783-1796

[3]

[Anonymous], 2002, AM J RESP CRIT CARE, V165, P277, DOI [DOI 10.1164/AJRCCM.165.2.ATS01, 10.1164/ajrccm.165.2.ats01]

[4]

Critical role for the chemokine MCP-1/CCR2 in the pathogenesis of bronchiolitis obliterans syndrome [J].

Belperio, JA ;

Keane, MP ;

Burdick, MD ;

Lynch, JP ;

Xue, YY ;

Berlin, A ;

Ross, DJ ;

Kunkel, SL ;

Charo, IF ;

Strieter, RM .

JOURNAL OF CLINICAL INVESTIGATION, 2001, 108 (04) :547-556

[5]

Integrin β1 signaling is necessary for transforming growth factor-β activation of p38MAPK and epithelial plasticity [J].

Bhowmick, NA ;

Zent, R ;

Ghiassi, M ;

McDonnell, M ;

Moses, HL .

JOURNAL OF BIOLOGICAL CHEMISTRY, 2001, 276 (50) :46707-46713

[6]

Pulmonary function and high-resolution CT findings in patients with an inherited form of pulmonary fibrosis, Hermansky-Pudlak syndrome, due to mutations in HPS-1 [J].

Brantly, M ;

Avila, NA ;

Shotelersuk, V ;

Lucero, C ;

Huizing, M ;

Gahl, WA .

CHEST, 2000, 117 (01) :129-136

[7]

PHYSIOLOGICAL CONSEQUENCES OF LOSS OF PLASMINOGEN-ACTIVATOR GENE-FUNCTION IN MICE [J].

CARMELIET, P ;

SCHOONJANS, L ;

KIECKENS, L ;

REAM, B ;

DEGEN, J ;

BRONSON, R ;

DEVOS, R ;

VANDENOORD, JJ ;

COLLEN, D ;

MULLIGAN, RC .

NATURE, 1994, 368 (6470) :419-424

[8]

Thrombin is a potent inducer of connective tissue growth factor production via proteolytic activation of protease-activated receptor-1 [J].

Chambers, RC ;

Leoni, P ;

Blanc-Brude, OP ;

Wembridge, DE ;

Laurent, GJ .

JOURNAL OF BIOLOGICAL CHEMISTRY, 2000, 275 (45) :35584-35591

[9]

REGULATION OF THE PROCOAGULANT ACTIVITY WITHIN THE BRONCHOALVEOLAR COMPARTMENT OF NORMAL HUMAN-LUNG [J].

CHAPMAN, HA ;

STAHL, M ;

ALLEN, CL ;

YEE, R ;

FAIR, DS .

AMERICAN REVIEW OF RESPIRATORY DISEASE, 1988, 137 (06) :1417-1425

[10]

CHAPMAN HA, 1986, AM REV RESPIR DIS, V133, P437

← 1 2 3 4 5 6 7 8 9 10 →