Rasmussen's encephalitis with concomitant cortical dysplasia: The role of GluR3

被引：30

作者：

Palmer, CA

Geyer, JD

Keating, JM

Gilliam, F

Kuzniecky, RI

Morawetz, RB

Bebin, EM

机构：

[1] Univ Alabama, Div Neuropathol, Birmingham, AL 35294 USA

[2] Univ Alabama, Dept Neurol, Birmingham, AL 35294 USA

[3] Univ Alabama, Div Neurosurg, Birmingham, AL 35294 USA

[4] Childrens Hosp, Dept Pediat, Birmingham, AL USA

来源：

EPILEPSIA | 1999年 / 40卷 / 02期

关键词：

Rasmussen's encephalitis; GluR3; receptor; cortical dysplasia; glutamate;

D O I：

10.1111/j.1528-1157.1999.tb02082.x

中图分类号：

R74 [神经病学与精神病学];

学科分类号：

摘要：

The role of the glutamate receptor GluR3 in Rasmussen's encephalitis is actively under investigation. Autoimmune processes with this receptor as thr target are currently theorized. We provide an additional case of pathologically proved Rasmussen's encephalitis (with concomitant cortical dysplasia) in the presence of antibodies against the GluR3 receptor.

引用

页码：242 / 247

页数：6

共 12 条

[1]

Adcock Jane E., 1997, Epilepsia, V38, P189

[2] EPILEPSIA PARTIALIS CONTINUA AND OTHER SEIZURES ARISING FROM THE PRECENTRAL GYRUS - HIGH-INCIDENCE IN PATIENTS WITH RASMUSSEN SYNDROME AND NEURONAL MIGRATION DISORDERS [J].