The RhD- trait in a white patient with the RhCCee phenotype attributed to a four-nucleotide deletion in the RHD gene

被引：23

作者：

Andrews, KT ^{[1
]}

Wolter, LC ^{[1
]}

Saul, A ^{[1
]}

Hyland, CA ^{[1
]}

机构：

[1] Queensland Inst Med Res, Australian Red Cross Blood Serv Queensland, Brisbane, Qld 4006, Australia

来源：

BLOOD | 1998年 / 92卷 / 05期

关键词：

D O I：

10.1182/blood.V92.5.1839.splL4_1839_1840

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

引用

页码：1839 / 1840

页数：2

共 12 条

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[3] Evidence of genetic diversity underlying Rh D-, weak D (D-u), and partial D phenotypes as determined by multiplex polymerase chain reaction analysis of the RHD gene
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[4] PRENATAL DETERMINATION OF FETAL RHD TYPE BY DNA AMPLIFICATION
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[J]. BLOOD REVIEWS, 1994, 8 (04) : 199 - 212
[7] Alteration of RH gene structure and expression in human dCCee and DCW-red blood cells: Phenotypic homozygosity versus genotypic heterozygosity
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[J]. BLOOD, 1996, 88 (06) : 2326 - 2333
[8] HYLAND CA, 1994, BLOOD, V84, P321
[9] MOLECULAR-CLONING AND PRIMARY STRUCTURE OF THE HUMAN BLOOD-GROUP RHD POLYPEPTIDE
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[J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1992, 89 (22) : 10925 - 10929
[10] The RHD gene is highly detectable in RhD-negative Japanese donors
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[J]. JOURNAL OF CLINICAL INVESTIGATION, 1997, 100 (02) : 373 - 379

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