Role of clinical pathologies in myocardial injury following ischaemia and reperfusion

被引：42

作者：

Galiñanes, M ^{[1
]}

Fowler, AG ^{[1
]}

机构：

[1] Univ Leicester, Glenfield Gen Hosp, Dept Cardiovasc Sci, Integrat Human Cardiovasc Physiol & Cardiac Surg, Leicester LE3 9QP, Leics, England

来源：

CARDIOVASCULAR RESEARCH | 2004年 / 61卷 / 03期

关键词：

reperfusion injury; diabetes; hypercholesterolaemia; hypertension; hypertrophy; heart failure;

D O I：

10.1016/j.cardiores.2003.11.028

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

The myocardial injury that occurs following a period of ischaemia and reperfusion is well recognised and the underlying mechanisms have been extensively investigated in healthy myocardial tissues; however, the influence of the risk factors associated with cardiovascular diseases on ischaemia/reperfusion injury remains unclear. In this article, the present knowledge on how clinical pathologies such as diabetes, hypercholesterolaemia, hypertension, myocardial hypertrophy and heart failure, may influence ischaemia/reperfusion injury of the heart and the potential mechanisms involved are reviewed. (C) 2003 European Society of Cardiology. Published by Elsevier B.V. All rights reserved.

引用

收藏

页码：512 / 521

页数：10

相关论文

共 176 条

[1] 5-YEAR ANGIOGRAPHIC FOLLOW-UP OF FACTORS ASSOCIATED WITH PROGRESSION OF CORONARY-ARTERY DISEASE IN THE CORONARY-ARTERY SURGERY STUDY (CASS) [J].

ALDERMAN, EL ;

CORLEY, SD ;

FISHER, LD ;

CHAITMAN, BR ;

FAXON, DP ;

FOSTER, ED ;

KILLIP, T ;

SOSA, JA ;

BOURASSA, MG ;

DOYLE, JT ;

MCKNEALLY, MF ;

MCILDUFF, JB ;

ODABASHIAN, H ;

OLDER, TM ;

RYAN, T ;

WEXLER, L ;

BARNER, HB ;

TYRAS, DH ;

CIPRIANO, PR ;

STINSON, EB ;

KENNEDY, JW ;

SOLOMON, R ;

PASSAMANI, ER ;

BERGER, RL ;

WEINER, D ;

GOULET, C ;

LESPERANCE, J ;

GRONDIN, CH ;

CASTONGUAY, Y ;

KAISER, G ;

MUDD, JG ;

WIENS, RD ;

CODD, JE ;

WILLMAN, VL ;

KOCH, FH ;

SILVERMAN, J ;

DAVIS, K ;

GILLESPIE, MJ ;

KRONMAL, R ;

SOPKO, G ;

ROBERTSON, T ;

FROMMER, P .

JOURNAL OF THE AMERICAN COLLEGE OF CARDIOLOGY, 1993, 22 (04) :1141-1154

[2] ASSOCIATION OF THE RENIN SODIUM PROFILE WITH THE RISK OF MYOCARDIAL-INFARCTION IN PATIENTS WITH HYPERTENSION [J].

ALDERMAN, MH ;

MADHAVAN, S ;

OOI, WL ;

COHEN, H ;

SEALEY, JE ;

LARAGH, JH .

NEW ENGLAND JOURNAL OF MEDICINE, 1991, 324 (16) :1098-1104

[3] CALCIUM OVERLOAD DURING REPERFUSION IS ACCELERATED IN ISOLATED HYPERTROPHIED RAT HEARTS [J].

ALLARD, MF ;

FLINT, JDA ;

ENGLISH, JC ;

HENNING, SL ;

SALAMANCA, MC ;

KAMIMURA, CT ;

ENGLISH, DR .

JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY, 1994, 26 (12) :1551-1563

[4] PREISCHEMIC GLYCOGEN REDUCTION OR GLYCOLYTIC INHIBITION IMPROVES POSTISCHEMIC RECOVERY OF HYPERTROPHIED RAT HEARTS [J].

ALLARD, MF ;

EMANUEL, PG ;

RUSSELL, JA ;

BISHOP, SP ;

DIGERNESS, SB ;

ANDERSON, PG .

AMERICAN JOURNAL OF PHYSIOLOGY, 1994, 267 (01) :H66-H74

[5]

ANDERSEN B, 1988, J LAB CLIN MED, V111, P275

[6] INCREASED ISCHEMIC-INJURY BUT DECREASED HYPOXIC INJURY IN HYPERTROPHIED RAT HEARTS [J].

ANDERSON, PG ;

ALLARD, MF ;

THOMAS, GD ;

BISHOP, SP ;

DIGERNESS, SB .

CIRCULATION RESEARCH, 1990, 67 (04) :948-959

[7] LOW-DENSITY LIPOPROTEINS INHIBIT ENDOTHELIUM-DEPENDENT RELAXATION IN RABBIT AORTA [J].

ANDREWS, HE ;

BRUCKDORFER, KR ;

DUNN, RC ;

JACOBS, M .

NATURE, 1987, 327 (6119) :237-239

[8] SARCOPLASMIC-RETICULUM GENE-EXPRESSION IN CARDIAC-HYPERTROPHY AND HEART-FAILURE [J].

ARAI, M ;

MATSUI, H ;

PERIASAMY, M .

CIRCULATION RESEARCH, 1994, 74 (04) :555-564

[9] Endothelium-derived nitric oxide and vascular physiology and pathology [J].

Arnal, JF ;

Dinh-Xuan, AT ;

Pueyo, M ;

Darblade, B ;

Rami, J .

CELLULAR AND MOLECULAR LIFE SCIENCES, 1999, 55 (8-9) :1078-1087

[10] Enhanced acute-phase response and oxidative stress in older adults with type II diabetes [J].

Arnalich, F ;

Hernanz, A ;

López-Maderuelo, D ;

Peña, JM ;

Camacho, J ;

Madero, R ;

Vázquez, JJ ;

Montiel, C .

HORMONE AND METABOLIC RESEARCH, 2000, 32 (10) :407-412

← 1 2 3 4 5 6 7 8 9 10 →