Modulation of protein kinase C in antitumor treatment

被引：68

作者：

Hofmann, J ^{[1
]}

机构：

[1] Univ Innsbruck, Inst Med Chem & Biochem, A-6020 Innsbruck, Austria

来源：

REVIEWS OF PHYSIOLOGY BIOCHEMISTRY AND PHARMACOLOGY | 2001年 / 142卷

关键词：

D O I：

10.1007/BFb0117491

中图分类号：

Q5 [生物化学]; Q7 [分子生物学];

学科分类号：

071010 ; 081704 ;

摘要：

引用

页码：1 / 96

页数：96

共 601 条

[1] HIV/gp120 and PMA/ionomycin induced apoptosis but not activation induced cell death require PKC for Fas-L upregulation [J].

Accornero, P ;

Radrizzani, M ;

Carè, A ;

Mattia, G ;

Chiodoni, C ;

Kurrle, R ;

Colombo, MP .

FEBS LETTERS, 1998, 436 (03) :461-465

[2] Both the catalytic and regulatory domains of protein kinase C chimeras modulate the proliferative properties of NIH 3T3 cells [J].

Acs, P ;

Wang, QMJ ;

Bogi, K ;

Marquez, AM ;

Lorenzo, PS ;

Biro, T ;

Szallasi, Z ;

Mushinski, JF ;

Blumberg, PM .

JOURNAL OF BIOLOGICAL CHEMISTRY, 1997, 272 (45) :28793-28799

[3]

Adams L. M., 1993, Proceedings of the American Association for Cancer Research Annual Meeting, V34, P410

[4]

Aebi S, 1996, CANCER RES, V56, P3087

[5]

AFTAB DT, 1994, ONCOL RES, V6, P59

[6] ANTISENSE EXPRESSION OF PROTEIN-KINASE C-ALPHA INHIBITS THE GROWTH AND TUMORIGENICITY OF HUMAN GLIOBLASTOMA CELLS [J].

AHMAD, S ;

MINETA, T ;

MARTUZA, RL ;

GLAZER, RI .

NEUROSURGERY, 1994, 35 (05) :904-908

[7]

AHMAD S, 1993, MOL PHARMACOL, V43, P858

[8]

AHMAD S, 1992, MOL PHARMACOL, V42, P1004

[9] EGR-1 induction is required for maximal radiosensitivity in A375-C6 melanoma cells [J].

Ahmed, MM ;

Venkatasubbarao, K ;

Fruitwala, SM ;

Muthukkumar, S ;

Wood, DP ;

Sells, SF ;

Mohiuddin, M ;

Rangnekar, VM .

JOURNAL OF BIOLOGICAL CHEMISTRY, 1996, 271 (46) :29231-29237

[10]

AHMED S, 1993, J BIOL CHEM, V268, P10709

← 1 2 3 4 5 6 7 8 9 10 →