Oxygen JNKies: Phosphatases overdose on ROS

被引:19
作者
Pham, CG [1 ]
Papa, S [1 ]
Bubici, C [1 ]
Zazzeroni, F [1 ]
Franzoso, G [1 ]
机构
[1] Univ Chicago, Ben May Inst Canc Res, Chicago, IL 60637 USA
关键词
D O I
10.1016/j.devcel.2005.03.005
中图分类号
Q2 [细胞生物学];
学科分类号
071009 [细胞生物学]; 090102 [作物遗传育种];
摘要
引用
收藏
页码:452 / 454
页数:3
相关论文
共 9 条
[1]
Induction of gadd45β by NF-κB downregulates pro-apoptotic JNK signalling [J].
De Smaele, E ;
Zazzeroni, F ;
Papa, S ;
Nguyen, DU ;
Jin, RG ;
Jones, J ;
Cong, R ;
Franzoso, G .
NATURE, 2001, 414 (6861) :308-313
[2]
Reactive oxygen species promote TNFα-induced death and sustained JNK activation by inhibiting MAP kinase phosphatases [J].
Kamata, H ;
Honda, S ;
Maeda, S ;
Chang, LF ;
Hirata, H ;
Karin, M .
CELL, 2005, 120 (05) :649-661
[3]
Stress-responsive protein kinases in redox-regulated apoptosis signaling [J].
Matsuzawa, A ;
Ichijo, H .
ANTIOXIDANTS & REDOX SIGNALING, 2005, 7 (3-4) :472-481
[4]
Ferritin heavy chain upregulation by NF-κB inhibits TNFα-induced apoptosis by suppressing reactive oxygen species [J].
Pham, CG ;
Bubici, C ;
Zazzeroni, F ;
Papa, S ;
Jones, J ;
Alvarez, K ;
Jayawardena, S ;
De Smaele, E ;
Cong, R ;
Beaumont, C ;
Torti, FM ;
Torti, SV ;
Franzoso, G .
CELL, 2004, 119 (04) :529-542
[5]
The p65/RelA subunit of NF-κB suppresses the sustained, antiapoptotic activity of Jun kinase induced by tumor necrosis factor [J].
Reuther-Madrid, JY ;
Kashatus, D ;
Chen, SQ ;
Li, X ;
Westwick, J ;
Davis, RJ ;
Earp, HS ;
Wang, CY ;
Baldwin, AS .
MOLECULAR AND CELLULAR BIOLOGY, 2002, 22 (23) :8175-8183
[6]
NF-κB inhibits TNF-induced accumulation of ROS that mediate prolonged MAPK activation and necrotic cell death [J].
Sakon, S ;
Xue, X ;
Takekawa, M ;
Sasazuki, T ;
Okazaki, T ;
Kojima, Y ;
Piao, JH ;
Yagita, H ;
Okumura, K ;
Doi, T ;
Nakano, H .
EMBO JOURNAL, 2003, 22 (15) :3898-3909
[7]
Inhibition of JNK activation through NF-κB target genes [J].
Tang, GL ;
Minemoto, Y ;
Dibling, B ;
Purcell, NH ;
Li, ZW ;
Karin, M ;
Lin, A .
NATURE, 2001, 414 (6861) :313-317
[8]
INK potentiates TNF-stimulated necrosis by increasing the production of cytotoxic reactive oxygen species [J].
Ventura, JJ ;
Cogswell, P ;
Flavell, RA ;
Baldwin, AS ;
Davis, RJ .
GENES & DEVELOPMENT, 2004, 18 (23) :2905-2915
[9]
Tumor necrosis factor signaling [J].
Wajant, H ;
Pfizenmaier, K ;
Scheurich, P .
CELL DEATH AND DIFFERENTIATION, 2003, 10 (01) :45-65