Neutrophil C5a receptor and the outcome in a rat model of sepsis

被引:66
作者
Guo, RF
Riedemann, NC
Bernacki, KD
Sarma, VJ
Laudes, IJ
Reuben, JS
Younkin, EM
Neff, TA
Paulauskis, JD
Zetoune, FS
Ward, PA
机构
[1] Univ Michigan, Sch Med, Dept Pathol, Ann Arbor, MI 48109 USA
[2] Pfizer Global Res & Dev, Dept Drug Safety Evaluat, Ann Arbor, MI 48105 USA
关键词
innate immunity; polymorphonuclear cells; complement;
D O I
10.1096/fj.03-0009fje
中图分类号
Q5 [生物化学]; Q7 [分子生物学];
学科分类号
071010 ; 081704 ;
摘要
Complement fragment 5a (C5a)-C5a receptor (C5aR) signaling plays an essential role in neutrophil innate immunity. Blockade of either the ligand or the receptor improves survival rates in experimental sepsis. In the current study, sepsis was induced in rats by cecal ligation/puncture. Early in sepsis C5aR content on neutrophils significantly dropped, reached the nadir at 24 h after onset of sepsis, and progressively elevated thereafter. Western-blot, RT-PCR, and confocal microscopy analyses revealed that the loss and re-expression of C5aR during sepsis might be due, at least in part, to the receptor internalization and reconstitution. The reduction and reconstitution of C5aR correlate with the loss and restoration of innate immune functions of blood neutrophils (chemotaxis and reactive oxygen species production), respectively. Quantitative measurements of C5aR on blood neutrophils are highly predictive of survival or death during sepsis. These data suggest that neutrophil C5aR content represents an essential component of an efficient defense system in sepsis and may serve as a prognostic marker for the outcome.
引用
收藏
页码:1889 / +
页数:17
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