Crigler-Najjar syndrome type II is inherited both as a dominant and as a recessive trait

被引:55
作者
Koiwai, O
Aono, S
Adachi, Y
Kamisako, T
Yasui, Y
Nishizawa, M
Sato, H
机构
[1] INST DEV RES,DEPT PERINATOL,KASUGAI,AICHI 48003,JAPAN
[2] KINKI UNIV,SCH MED,DEPT INTERNAL MED 2,OSAKA 589,JAPAN
[3] SHIGA UNIV MED SCI,DEPT BIOL,SHIGA 52021,JAPAN
关键词
D O I
10.1093/hmg/5.5.645
中图分类号
Q5 [生物化学]; Q7 [分子生物学];
学科分类号
071010 ; 081704 ;
摘要
Crigler-Najjar syndrome type II (CN-II) is caused by a severely reduced hepatic activity of bilirubin UDP-glucuronosyltransferase (UGT). Recently, by the analysis of the genetic background of CN-II patients, it has been clarified that the patients carry homozygous missense mutations or nonsense plus missense mutations on the gene for UGT, and CN-II was inherited as an autosomal recessive trait. We encountered a new case which had a nonsense mutation caused by a single nucleotide substitution on one allele. This indicates that CN-II is also inherited as a dominant trait as well as a recessive trait. Expression study in vitro strongly suggests that the disease in this case is caused by a dominant negative mutation by forming a heterologous subunit structure.
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收藏
页码:645 / 647
页数:3
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