Synaptic plasticity in ischemia:: Role of NMDA receptors

[1] MECHANISM OF ARACHIDONIC-ACID LIBERATION DURING ISCHEMIA IN GERBIL CEREBRAL-CORTEX [J]. JOURNAL OF NEUROCHEMISTRY, 1987, 48 (02) : 503 - 509

[2] OXYGEN FREE-RADICALS REGULATE NMDA RECEPTOR FUNCTION VIA A REDOX MODULATORY SITE [J]. NEURON, 1990, 5 (06) : 841 - 846

[3] ENHANCED CALCIUM-UPTAKE BY CA1 PYRAMIDAL CELL DENDRITES IN THE POSTISCHEMIC PHASE DESPITE SUBNORMAL EVOKED FIELD POTENTIALS - EXCITATORY AMINO-ACID RECEPTOR DEPENDENCY AND RELATIONSHIP TO NEURONAL DAMAGE [J]. JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM, 1992, 12 (05) : 773 - 783

[4] CALCIUM-UPTAKE EVOKED BY ELECTRICAL-STIMULATION IS ENHANCED POSTISCHEMICALLY AND PRECEDES DELAYED NEURONAL DEATH IN CA1 OF RAT HIPPOCAMPUS - INVOLEMENT OF N-METHYL-D-ASPARTATE RECEPTORS [J]. JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM, 1988, 8 (06) : 799 - 807

[5] Expression of LTP by AMPA and/or NMDA receptors is determined by the extent of NMDA receptors activation during the tetanus [J]. JOURNAL OF NEUROPHYSIOLOGY, 1995, 74 (06) : 2349 - 2357

[6] NEURONAL DAMAGE AND CALCIUM ACCUMULATION FOLLOWING REPEATED BRIEF CEREBRAL-ISCHEMIA IN THE GERBIL [J]. BRAIN RESEARCH, 1990, 528 (01) : 114 - 122

[7] ABSENCE OF ELECTROGRAPHIC SEIZURES AFTER TRANSIENT FOREBRAIN ISCHEMIA IN THE MONGOLIAN GERBIL [J]. BRAIN RESEARCH, 1989, 476 (01) : 174 - 178

[8] Ben-Ari Y., 1994, Society for Neuroscience Abstracts, V20, P1517

[9] PROTEIN-KINASE-C MODULATION OF NMDA CURRENTS - AN IMPORTANT LINK FOR LTP INDUCTION [J]. TRENDS IN NEUROSCIENCES, 1992, 15 (09) : 333 - 339

[10] ELEVATION OF THE EXTRACELLULAR CONCENTRATIONS OF GLUTAMATE AND ASPARTATE IN RAT HIPPOCAMPUS DURING TRANSIENT CEREBRAL-ISCHEMIA MONITORED BY INTRACEREBRAL MICRODIALYSIS [J]. JOURNAL OF NEUROCHEMISTRY, 1984, 43 (05) : 1369 - 1374

[1] MECHANISM OF ARACHIDONIC-ACID LIBERATION DURING ISCHEMIA IN GERBIL CEREBRAL-CORTEX [J]. JOURNAL OF NEUROCHEMISTRY, 1987, 48 (02) : 503 - 509

[2] OXYGEN FREE-RADICALS REGULATE NMDA RECEPTOR FUNCTION VIA A REDOX MODULATORY SITE [J]. NEURON, 1990, 5 (06) : 841 - 846

[3] ENHANCED CALCIUM-UPTAKE BY CA1 PYRAMIDAL CELL DENDRITES IN THE POSTISCHEMIC PHASE DESPITE SUBNORMAL EVOKED FIELD POTENTIALS - EXCITATORY AMINO-ACID RECEPTOR DEPENDENCY AND RELATIONSHIP TO NEURONAL DAMAGE [J]. JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM, 1992, 12 (05) : 773 - 783

[4] CALCIUM-UPTAKE EVOKED BY ELECTRICAL-STIMULATION IS ENHANCED POSTISCHEMICALLY AND PRECEDES DELAYED NEURONAL DEATH IN CA1 OF RAT HIPPOCAMPUS - INVOLEMENT OF N-METHYL-D-ASPARTATE RECEPTORS [J]. JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM, 1988, 8 (06) : 799 - 807

[5] Expression of LTP by AMPA and/or NMDA receptors is determined by the extent of NMDA receptors activation during the tetanus [J]. JOURNAL OF NEUROPHYSIOLOGY, 1995, 74 (06) : 2349 - 2357

[6] NEURONAL DAMAGE AND CALCIUM ACCUMULATION FOLLOWING REPEATED BRIEF CEREBRAL-ISCHEMIA IN THE GERBIL [J]. BRAIN RESEARCH, 1990, 528 (01) : 114 - 122

[7] ABSENCE OF ELECTROGRAPHIC SEIZURES AFTER TRANSIENT FOREBRAIN ISCHEMIA IN THE MONGOLIAN GERBIL [J]. BRAIN RESEARCH, 1989, 476 (01) : 174 - 178

[8] Ben-Ari Y., 1994, Society for Neuroscience Abstracts, V20, P1517

[9] PROTEIN-KINASE-C MODULATION OF NMDA CURRENTS - AN IMPORTANT LINK FOR LTP INDUCTION [J]. TRENDS IN NEUROSCIENCES, 1992, 15 (09) : 333 - 339

[10] ELEVATION OF THE EXTRACELLULAR CONCENTRATIONS OF GLUTAMATE AND ASPARTATE IN RAT HIPPOCAMPUS DURING TRANSIENT CEREBRAL-ISCHEMIA MONITORED BY INTRACEREBRAL MICRODIALYSIS [J]. JOURNAL OF NEUROCHEMISTRY, 1984, 43 (05) : 1369 - 1374