Hypermethylation of p15INK4B gene in a patient with acute myelogenous leukemia evolved from paroxysmal nocturnal hemoglobinuria

被引：6

作者：

Uchida, T ^{[1
]}

Ohashi, H

Kinoshita, T

Saito, H

Taguchi, R

Hotta, T

Murate, T

机构：

[1] Nagoya Univ, Sch Med, Dept Internal Med 1, Nagoya, Aichi 466, Japan

[2] Nagoya City Univ, Fac Pharmaceut Sci, Nagoya, Aichi 467, Japan

[3] Tokai Univ, Sch Med, Dept Internal Med 4, Isehara, Kanagawa 25911, Japan

[4] Nagoya Univ, Sch Hlth Sci, Nagoya, Aichi 466, Japan

来源：

BLOOD | 1998年 / 92卷 / 08期

关键词：

D O I：

10.1182/blood.V92.8.2981

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

引用

页码：2981 / 2983

页数：3

共 7 条

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[4] Impaired hematopoiesis in paroxysmal nocturnal hemoglobinuria/aplastic anemia is not associated with a selective proliferative defect in the glycosylphosphatidylinositol-anchored protein-deficient clone [J].

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[5]

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[6] Methylation of the p15INK4a gene in myelodysplastic syndromes is frequent and acquired during disease progression [J].

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BLOOD, 1998, 91 (08) :2985-2990

[7] Hypermethylation of the p15(INK4B) gene in myelodysplastic syndromes [J].

Uchida, T ;

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BLOOD, 1997, 90 (04) :1403-1409

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