Blood catalase deficiency and diabetes in Hungary

被引：40

作者：

Góth, L

Lenkey, A

Bigler, WN

机构：

[1] Univ Debrecen, Dept Clin Biochem & Mol Pathol, H-4012 Debrecen, Hungary

[2] Univ Debrecen, Dept Clin Analyt Chem, H-4012 Debrecen, Hungary

[3] San Francisco State Univ, Ctr Biomed Lab Sci, San Francisco, CA 94132 USA

来源：

DIABETES CARE | 2001年 / 24卷 / 10期

关键词：

D O I：

10.2337/diacare.24.10.1839

中图分类号：

R5 [内科学];

学科分类号：

1002 [临床医学]; 100201 [内科学];

摘要：

引用

页码：1839 / 1840

页数：2

共 18 条

[1]

[Anonymous], 1995, METABOLIC MOL BASES

[2]

GAETANI GF, 1996, BLOOD, V87, P1569

[3]

Hereditary catalase deficiencies and increased risk of diabetes [J].

Góth, L ;

Eaton, JW .

LANCET, 2000, 356 (9244) :1820-1821

[4]

Goth L, 1996, CLIN CHEM, V42, P341

[5]

2 CASES OF ACATALASEMIA IN HUNGARY [J].

GOTH, L .

CLINICA CHIMICA ACTA, 1992, 207 (1-2) :155-158

[6]

Góth L, 2000, CLIN CHEM, V46, P564

[7]

HAUSEN LL, 1999, J BIOL CHEM, V274, P25078

[8]

Catalase deficiency, diabetes, and mitochondrial function [J].

Heales, SJR .

LANCET, 2001, 357 (9252) :314-314

[9]

Effect of superoxide dismutase, catalase, chelating agents, and free radical scavengers on the toxicity of alloxan to isolated pancreatic islets in vitro [J].

Jörns, A ;

Tiedge, M ;

Lenzen, S ;

Munday, R .

FREE RADICAL BIOLOGY AND MEDICINE, 1999, 26 (9-10) :1300-1304

[10]

Direct evidence for catalase as the predominant H2O2-removing enzyme in human erythrocytes [J].

Mueller, S ;

Riedel, HD ;

Stremmel, W .

BLOOD, 1997, 90 (12) :4973-4978

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