Involvement of jasmonic acid in elicitor-induced phytoalexin production in suspension-cultured rice cells

被引:197
作者
Nojiri, H
Sugimori, M
Yamane, H
Nishimura, Y
Yamada, A
Shibuya, N
Kodama, O
Murofushi, N
Omori, T
机构
[1] UNIV TOKYO,BIOTECHNOL RES CTR,BUNKYO KU,TOKYO 113,JAPAN
[2] NATL INST AGROBIOL RESOURCES,DEPT CELL BIOL,TSUKUBA,IBARAKI 305,JAPAN
[3] IBARAKI UNIV,LAB BIOORGAN & PESTICIDE CHEM,AMI,IBARAKI 30003,JAPAN
[4] UNIV TOKYO,DEPT APPL BIOL CHEM,BUNKYO KU,TOKYO 113,JAPAN
关键词
D O I
10.1104/pp.110.2.387
中图分类号
Q94 [植物学];
学科分类号
071001 ;
摘要
It has been suggested that jasmonic acid (JA) could be an integral part of a general signal transduction system regulating inducible defense genes in plants. It was reported that treatment with an elicitor (N-acetylchitoheptaose) induced production of phytoalexin in suspension-cultured rice (Oryza sativa L.) cells. In this study, the role of JA in the induction of phytoalexin production by N-acetylchitoheptaose was investigated. Exogenously applied (+/-)-JA (10(-4) M) clearly induced the production of momilactone A, a major phytoalexin, in suspension-cultured rice cells. On the other hand, in rice cells treated with N-acetylchitoheptaose, endogenous JA was rapidly and transiently accumulated prior to accumulation of momilactone A. Treatment with ibuprofen, an inhibitor of JA biosynthesis, reduced production of momilactone A in the cells treated with N-acetylchitoheptaose, but the addition of (+/-)-JA increased production of momilactone A to levels higher than those in the elicited rice cells. These results strongly suggest that JA functions as a signal transducer in the induction of biosynthesis of momilactone A by N-acetylchitoheptaose in suspension-cultured rice cells.
引用
收藏
页码:387 / 392
页数:6
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